Reduction of cell viability via FINs is (partially) reversed by deferoxamine mesylate and ferrostatin-1. by Dino Bekric (16475280)

“…<p>Cells were treated with 1 μM brequinar, 10 μM FINO2, 50 μM iFSP1, 50 μM IKE and 10 μM RSL3 with 20 μM of deferoxamine mesylate, ferrostatin-1, necrostatin-1 and Z-VAD-FMK. (A) Cell viability data is shown as mean values +/- SEM of n = 3 biological replicates of CCC-5 cells with FINs (light grey) and FINS + cell death inhibitors (dark grey) treatment for 24 h. …”

Table_1_Prevalence and WGS-based characteristics of MRSA isolates in hospitals in Shanghai, China.XLS by Hongzhi Zhang (425117)

“…<p>Methicillin-resistant Staphylococcus aureus (MRSA) isolates remain a serious threat to global health despite a decrease in MRSA infections since 2005. …”

Complementary interactions of cysteine and Fe(II) with 6PG. by Gabriel Piedrafita (155328)

“…Individual measurements (dots) and mean (horizontal line) from <i>N</i> ≥ 4 experiments (see <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3001468#pbio.3001468.s010" target="_blank">S1 Data</a>). 6PG, 6-phosphogluconic acid; DOSY, diffusion ordered spectroscopy; R5P, ribose-5-phosphate.…”

Loss of <i>bmm</i> function in neurons with an independent RNAi line and additional neuronal GAL4 line affects whole-body triglyceride breakdown in males. by Lianna W. Wat (8339454)

“…(E) In dissected brains from <i>elav>UAS-bmm-RNAi</i> males, we found that <i>bmm</i> transcript levels were undetectable in three out of four samples, whereas we observed amplification at a higher cycle number in three <i>elav>+</i> samples. (§) Because of this dramatic decrease in <i>bmm</i> transcript levels in most <i>elav>UAS-bmm-RNAi</i> samples, we were unable to perform a statistical analysis to quantify this effect. …”

Table_1_Estimation of Cerebral Hemodynamics and Oxygenation During Various Intensities of Rowing Exercise: An NIRS Study.docx by Mikio Hiura (12176669)

“…</p>Methods<p>Eleven rowers, consisting out of six athletes and five recreational rowers [two female; age, 27 ± 9 years; height, 171 ± 7 cm, body mass, 67 ± 9 kg; V.O_2peak, 53.5 ± 6.5 mL min^–1 kg^–1] rowed a 13-min session separated by 10 and 3 min, at 70 (Ex_70%) and 80% of V.O_2peak (Ex_80%), respectively, on a rowing ergometer, followed by three sessions of 1-min supramaximal exercise (ExSp). …”

Table_1_Comparison of prepartum blood parameters in dairy cows with postpartum ketosis and new risk prediction candidates.DOCX by Woojae Choi (14775910)

“…Primiparous KET showed lower carboxylated osteocalcin (cOC) levels and a significant decrease after parturition. In multiparous KET (n = 96) before parturition, lower neutrophil (Neu) in BW–5, higher hemoglobin (HGB) in BW–5, higher MCV in BW–5 and BW–1, higher MCH in BW–5, lower total cholesterol (TC) in BW–5, higher triglyceride (TG) in BW–3, higher NEFA in BW–1, higher glucose (Glu) in BW–3, lower γ-glutamyl transferase (GGT) in BW–5, lower inorganic phosphate (iP) in BW–3, and higher body condition score (BCS) in BW–5 and BW–3 were significant compared with CON. …”

Table_1_Comparison of prepartum blood parameters in dairy cows with postpartum ketosis and new risk prediction candidates.DOCX by Woojae Choi (14775910)

“…Primiparous KET showed lower carboxylated osteocalcin (cOC) levels and a significant decrease after parturition. In multiparous KET (n = 96) before parturition, lower neutrophil (Neu) in BW–5, higher hemoglobin (HGB) in BW–5, higher MCV in BW–5 and BW–1, higher MCH in BW–5, lower total cholesterol (TC) in BW–5, higher triglyceride (TG) in BW–3, higher NEFA in BW–1, higher glucose (Glu) in BW–3, lower γ-glutamyl transferase (GGT) in BW–5, lower inorganic phosphate (iP) in BW–3, and higher body condition score (BCS) in BW–5 and BW–3 were significant compared with CON. …”

Table_1_Comparison of prepartum blood parameters in dairy cows with postpartum ketosis and new risk prediction candidates.DOCX by Woojae Choi (14775910)

“…Primiparous KET showed lower carboxylated osteocalcin (cOC) levels and a significant decrease after parturition. In multiparous KET (n = 96) before parturition, lower neutrophil (Neu) in BW–5, higher hemoglobin (HGB) in BW–5, higher MCV in BW–5 and BW–1, higher MCH in BW–5, lower total cholesterol (TC) in BW–5, higher triglyceride (TG) in BW–3, higher NEFA in BW–1, higher glucose (Glu) in BW–3, lower γ-glutamyl transferase (GGT) in BW–5, lower inorganic phosphate (iP) in BW–3, and higher body condition score (BCS) in BW–5 and BW–3 were significant compared with CON. …”

Intracellular LF82 multiplication requires stress responses. by Gaëlle Demarre (249248)

“…Horizontal lines indicate viability decrease by 2, 5 and 10 fold compared to WT LF82. Data were analyzed using a Student’s <i>t</i> test: * <i>P</i> <0.05, ** <i>P</i> <0.01, *** <i>P</i> <0.001, **** <i>P</i> <0.0001 (black stars correspond to mutant vs WT, blue stars correspond to mutant vs its complementation).…”

Supplementary file 1_Global burden and trends of adverse effects of medical treatment, 1990–2021: an analysis from the global burden of disease study 2021.docx by Keqing Zhang (326894)

“…Both age-standardized mortality (ASMR) and DALYs rates (ASDR) showed a gradual decline during the study period, but they still carried a heavy burden in the low SDI regions (2021 ASDR: 3.71 [95% UI: 2.90 to 5.68] per 100,000 persons-year; 2021 ASR for DALYs: 150.37 [95% UI: 109.08 to 215.24] per 100,000 persons-year). …”

Image1_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.TIF by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Table1_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.XLSX by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Table2_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.XLSX by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

DataSheet1_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.xlsx by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Image2_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.TIF by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Image3_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.TIF by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Image4_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.TIF by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Image6_Enteroendocrine Cell Formation Is an Early Event in Pancreatic Tumorigenesis.TIF by Leah R. Caplan (12464541)

“…<p>Pancreatic ductal adenocarcinoma (PDAC) is a devastating disease with a 5-year survival rate of only 11%, due, in part, to late diagnosis, making the need to understand early events in tumorigenesis critical. …”

Image1_Morphological, electrophysiological, and molecular alterations in foetal noncompacted cardiomyopathy induced by disruption of ROCK signalling.TIF by David Sedmera (636625)

“…The cause of the early noncompaction at E15.5 was attributed to a decrease in proliferation in the developing ventricular wall. …”

Data_Sheet_1_Homeostatic regulation of extracellular signal-regulated kinase 1/2 activity and axonal Kv7.3 expression by prolonged blockade of hippocampal neuronal activity.pdf by Brian C. Baculis (13165416)

“…However, whether a decrease in K_v7.3 expression occurs at the AIS during homeostatic scaling of intrinsic excitability and what signaling pathway reduces KCNQ3 transcript upon prolonged activity blockade remain unknown. …”