Drp1^SD overexpression partially alleviates the differentiation defect seen in <i>notch</i> RNAi expressing type II NB lineages. by Dnyanesh Dubal (12085942)

Expression of mRFP does not perturb the severity of <i>opa1</i> RNAi and <i>marf</i> RNAi phenotypes. by Dnyanesh Dubal (12085942)

Downregulation of Opa1 and Marf does affect type II NB numbers but affect differentiated cells in lineage. by Dnyanesh Dubal (12085942)

Analysis of proliferation in NBs depleted of Opa1 and Marf. by Dnyanesh Dubal (12085942)

Depletion of Opa1 and Marf does not cause increase in the cleaved Caspase 3 levels and apoptosis in the brain. by Dnyanesh Dubal (12085942)

Depletion of mitochondrial fusion proteins Opa1 and Marf result in mitochondrial fragmentation. by Dnyanesh Dubal (12085942)

Proliferation defects in Opa1 depleted type II NB lineages are suppressed by overexpression of Drp1^SD. by Dnyanesh Dubal (12085942)

“…<p>A,C-E: Representative confocal images of type II NB lineages show reduced pH3 positive mature INPs (A) in <i>opa1</i> RNAi while increase in <i>marf</i> RNAi expressed with <i>pnt</i>-Gal4, mCD8-GFP in the type II NB lineage. …”

Inhibition of ABCG2 decreases SP cells, but not sphere-forming ability. by Noriko Kawai (6694010)

Depletion of Opa1 shows increased ROS levels in the type II NB lineage. by Dnyanesh Dubal (12085942)

Subcellular levels of p53 protein in hTCEpi-TR cell lines. by Vindhya Koppaka (845151)

NOX4 depletion decreases cAMP concentration and AQP2 expression in a phosphodiesterase-dependent manner in mpkCCD_cl4 cells. by Eric Féraille (513934)

Opa1 and Marf depletion leads to fragmented mitochondria and overexpression of Drp1^SD leads to clustered mitochondria in type II NBs. by Dnyanesh Dubal (12085942)

Suppression of endogenous human TRAM expression decreases R848 mediated <i>CCL5</i> and <i>IFN-β</i>, but not <i>TNF-α</i> expression. by Enda Shevlin (628477)

a-c: Bone marrow (BM) EPO receptor (EPO-R) decreases in uremic anemia rats. by Daniel Landau (440830)

“…<p>BM EPO-R protein (depicted as percentage of actin-corrected concentration) is unchanged in 7 day-bled rats Vs controls (Fig. 4a). Contrary to that BM EPO-R protein decreases in uremic (4 weeks after 5/6 nephrectomy) rats (Fig. 4b). …”

Decreased transcription of <i>prs</i> results in decreased growth. by Javiera Norambuena (137228)

Molecular insights in the pathogenesis of classical Ehlers-Danlos syndrome from transcriptome-wide expression profiling of patients’ skin fibroblasts by Nicola Chiarelli (756982)

“…<div><p>Classical Ehlers-Danlos syndrome (cEDS) is a dominant inherited connective tissue disorder mainly caused by mutations in the <i>COL5A1</i> and <i>COL5A2</i> genes encoding type V collagen (COLLV), which is a fibrillar COLL widely distributed in a variety of connective tissues. cEDS patients suffer from skin hyperextensibility, abnormal wound healing/atrophic scars, and joint hypermobility. …”

Decreased oscillatory flow decreases <i>klf2a</i> expression. by Julien Vermot (257990)

“…<p>(A) RFF is decreased by alterations in heart rate. …”

Interaction of eIF5a with mitochondrial dynamics–related proteins. by Dong Ma (584680)

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eIF5a interacts with Mfn1 and preserves mitochondrial integrity. by Dong Ma (584680)

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NAC suppresses Ang II–induced mitochondrial fission in mouse VSMCs. by Dong Ma (584680)

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