Showing 11,901 - 11,920 results of 103,840 for search '(( 5 g decrease ) OR ( 5 ((we decrease) OR (((nn decrease) OR (a decrease)))) ))', query time: 2.08s Refine Results
  1. 11901

    Evaluating the Reduced Hydrophobic Taste Sensor Response of Dipeptides by Theasinensin A by Using NMR and Quantum Mechanical Analyses by Jian Guo (191157)

    Published 2016
    “…Quantum mechanical calculations and rotating frame nuclear Overhauser effect-NMR spectroscopy provided configuration information on the geometry of the complex that Trp-Leu formed with TSA (1:1 complex) with a Δ<i>G</i> energy of –8.7 kJ/mol. A sensor analysis using artificial-lipid membranes demonstrated that the changes in membrane potential of 1 mM Trp-Leu (21.8 ± 1.3 mV) and Leu-Trp (5.3 ± 0.9 mV) were significantly (<i>P</i> < 0.001) reduced by 1 mM TSA (Trp-Leu, 13.1 ± 2.4 mV; Leu-Trp, 3.5 ± 0.5 mV; TSA alone, 0.2 ± 0.01 mV), indicating the effective suppression of hydrophobicity of dipeptides by TSA-formed complex.…”
  2. 11902

    The expression of Atpv1c1 was efficiently knocked down by AAV-sh-<i>Atp6v1c1</i>. by Sheng Li (107261)

    Published 2015
    “…<p>(A-G) M-CSF/RANKL was applied for 3 days to allow differentiation of osteoclasts, which were then transduced with AAV-sh-Atp6v1c1, AAV-sh-luc-YFP, or untreated (mock). …”
  3. 11903

    Effect of Na<sub>V</sub>1.1 epileptogenic mutations on the firing of GABAergic neurons: Input-output relationship and action potential amplitude. by Louisiane Lemaire (5044049)

    Published 2021
    “…<p><b>A</b>: Model simulations. We applied to the GABAergic neuron at rest 2.5 s long excitatory external inputs of conductances <i>g</i><sub>D,i</sub>, for the default parameters (wild-type), or for a reduced sodium fast-inactivating maximal conductance <i>g</i><sub>Na,FI,i</sub> (epileptogenic mutations). …”
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  11. 11911

    Downregulation of SIRT1 signaling underlies hepatic autophagy impairment in glycogen storage disease type Ia by Jun-Ho Cho (4053676)

    Published 2017
    “…Consistently, defective autophagy in G6Pase-α-deficient liver is characterized by attenuated expressions of autophagy components, increased acetylation of ATG5 and ATG7, decreased conjugation of ATG5 and ATG12, and reduced autophagic flux. …”
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