Correlates of travel mode to school stratified by country in multivariable analyses. by Lucy-Joy Wachira (11985899)

Subjects:

Image_1_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.TIF by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Image_2_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.TIF by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Table_2_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.docx by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Table_1_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.docx by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Table_3_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.docx by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Data_Sheet_1_Evaluating Uncertainties in Reconstructing the Pre-eutrophic State of the North Sea.docx by Christoph Stegert (10427723)

“…For the reconstruction of the historical state, we use two coupled physical-biogeochemical model systems: ECOHAM on a 20-km grid for the European shelf and GPM on a high-resolution (1.5–4.5 km) grid for the Southern North Sea. …”

Long non-coding RNA Opa interacting protein 5-antisense RNA 1 binds to micorRNA-34a to upregulate oncogenic PD-L1 in non-small cell lung cancer by Xinwei Qiao (488240)

“…In addition, inhibition of OIP5-AS1 and overexpression of miR-34a decreased the expression levels of PD-L1, and miR-34a significantly blocked the role of overexpression of OIP5-AS1. …”

Snail infection prevalence in 0.38 ppt, 1.9 ppt, 3.8 ppt, and 5.7 ppt salinity treatments after exposure to <i>Schistosoma mansoni</i> miracidia. by Ao Yu (1348512)

Procedure to capture digital images of the body color of ornamental shrimp. by Wei-Wei Hou (3405947)

Molecular Insight into the Growth of Hydrogen and Methane Binary Hydrates by Zhengcai Zhang (2235856)

“…Moreover, with increasing temperature, the occupancy of 5¹² and 5¹²6⁴ cages by H₂ decreases, and inversely, the occupancy of cages by CH₄ increases when the temperature is above 240 K. …”

Effect of HCQ on cytokines after a 12-hour stimulation with 8μg/mL HPV (human papilloma virus) peptide pool. by Monika M. Kowatsch (11722292)

DataSheet1_The tumor suppressor 5A2, a synthetic miR-7-5p mimic, targets oncogenic and metabolic pathways, as revealed by transcriptome-wide analysis.PDF by Marion T. J. van den Bosch (16509735)

“…Interestingly, Ingenuity Pathway Analysis revealed a new role for 5A2 in metabolic pathways. Validation experiments in vitro showed that 5A2 reduced the expression of key glycolysis and glutaminolysis enzymes, leading to a decrease in glycolysis, lactate secretion and intracellular glutamate availability. …”

TKI increases parkin ubiquitination and interaction with Beclin-1. by Irina Lonskaya (429729)

Image_2_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”

Image_3_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”

Image_3_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”

Image_1_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”

Image_1_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”

Image_2_Tumor Suppressor miR-184 Enhances Chemosensitivity by Directly Inhibiting SLC7A5 in Retinoblastoma.TIF by Tian-Geng He (7870724)

“…Molecular studies revealed that miR-184-decreased phosphorylation status of known DNA damage repair sensors of the ATR/ATM pathways and induced persistent formation of γH2AX foci depend on targeting SLC7A5, leading to persistent DNA damage. …”