Explanatory power of risk measures in bearish sample. by Mihály Ormos (697939)

Predicting power in short periods out of sample. by Mihály Ormos (697939)

Mst1 knockout skewed macrophage polarization toward a proinflammatory and profibrotic phenotype with concomitant down-regulation of CD36. by Jianyang Li (461703)

Climate complexity in the migratory cycle of <i>Ammodramus bairdii</i> by Alexander Peña-Peniche (5687786)

“…A continuous and alarming decrease of its populations has been observed over the last 50 years, and studying its seasonal distribution and associated climatic niches could help improve strategies for its conservation. …”

Explanatory power of risk measures in long term by diversification. by Mihály Ormos (697939)

Secondary attack rates among 1407 close contacts based on case-patient^a characteristics, their own characteristics, and exposure setting. by Joseph Walker (4341838)

Blockade of IFN<i>γ</i> production rescues the T_reg cell homeostatic defect and alleviates systemic autoimmunity in <i>Foxp3</i>^<i>Cre</i><i>miR-142</i><sup><... by Wei-Le Wang (133402)

Redesigning BinQ catalytic specificity for the human CCR5 gene and AAVS1 safe harbor locus. by Mark C. Wallen (800888)

“…(<b>B, C)</b> Selection of BinQ mutants that recombine the symmetrical versions of the “left” (blue) and “right” (red) <b>(B)</b> AAVS1 and <b>(C)</b> CCR5 target sites. Asterisks indicate selection steps in which incubation time was decreased from 16 to 4 hr. …”

PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

HCV elicits differential expression of circRNAs. by Tzu-Chun Chen (787255)

Average Φ and surprisal over trial time for the easy task (black), the hard task (blue) and perfect animats in the hard task (red). by Christoffer Lundbak Olesen (17262937)

Predicting Dinitrogen Activation and Coupling with Carbon Dioxide and Other Small Molecules by Methyleneborane: A Combined DFT and Machine Learning Study by Feiying You (22119041)

Mendelian randomization results with COVID-19. by Alish B. Palmos (5857379)

Schematic diagram of STTFP model. by Guozhu Sui (21672798)

Expression of CRYAB in normal and tumor tissue samples across different cancer types. by Rasool Saghaleyni (10526301)

Flowchart style diagram of methods to sex GD 16.5 to GD 18.0 mice using the external genitalia. by Laura B. Murdaugh (5059100)

“…<p>Steps are listed in decreasing order of effectiveness for mice at GD 17.0–17.5; Step 1 is the most effective, and step 4 is the least effective. …”