Showing 601 - 620 results of 100,884 for search '(( 5 we decrease ) OR ( 5 ((step decrease) OR (((teer decrease) OR (a decrease)))) ))', query time: 1.66s Refine Results
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    DataSheet2_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

    Published 2022
    “…Since WNT5A-responsive mechanisms on eMSC are still uncertain, we hypothesize that the WNT ligand–WNT5A works to activate WNT/β-catenin signaling through binding to Frizzled receptors (FZDs) and co-receptor low-density lipoprotein receptor-related protein 5 (LRP5). …”
  12. 612

    DataSheet1_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

    Published 2022
    “…Since WNT5A-responsive mechanisms on eMSC are still uncertain, we hypothesize that the WNT ligand–WNT5A works to activate WNT/β-catenin signaling through binding to Frizzled receptors (FZDs) and co-receptor low-density lipoprotein receptor-related protein 5 (LRP5). …”
  13. 613

    DataSheet2_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

    Published 2022
    “…Since WNT5A-responsive mechanisms on eMSC are still uncertain, we hypothesize that the WNT ligand–WNT5A works to activate WNT/β-catenin signaling through binding to Frizzled receptors (FZDs) and co-receptor low-density lipoprotein receptor-related protein 5 (LRP5). …”
  14. 614

    DataSheet1_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

    Published 2022
    “…Since WNT5A-responsive mechanisms on eMSC are still uncertain, we hypothesize that the WNT ligand–WNT5A works to activate WNT/β-catenin signaling through binding to Frizzled receptors (FZDs) and co-receptor low-density lipoprotein receptor-related protein 5 (LRP5). …”
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    Applying antibody-sensitive hypervariable region 1-deleted hepatitis C virus to the study of escape pathways of neutralizing human monoclonal antibody AR5A by Rodrigo Velázquez-Moctezuma (3776023)

    Published 2017
    “…Culturing J6/JFH1<sub>ΔHVR1</sub> (genotype 2a), for which fitness was decreased by L665W, with AR5A generated AR5A-resistant viruses with the substitutions I345V, L665S, and S680T, which we introduced into J6/JFH1 and J6/JFH1<sub>ΔHVR1</sub>. …”
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