Showing 11,941 - 11,960 results of 100,240 for search '(( 5 wt decrease ) OR ( 5 ((teer decrease) OR (((mean decrease) OR (a decrease)))) ))', query time: 1.44s Refine Results
  1. 11941
  2. 11942
  3. 11943
  4. 11944

    Image5_A role for TRPC3 in mammalian testis development.jpg by Zhenhua Ming (5179547)

    Published 2024
    “…Trpc3 is also expressed in endothelial cells and Pyr3-treated E11.5 XY mouse gonads showed a loss of the coelomic blood vessel due to increased apoptosis of endothelial cells. …”
  5. 11945

    Image5_A role for TRPC3 in mammalian testis development.TIF by Zhenhua Ming (5179547)

    Published 2024
    “…Trpc3 is also expressed in endothelial cells and Pyr3-treated E11.5 XY mouse gonads showed a loss of the coelomic blood vessel due to increased apoptosis of endothelial cells. …”
  6. 11946
  7. 11947

    Generation of WT and PPT1 KO palmitome and proteome expands the repertoire of known palmitoylated proteins in the brain. by Erica L. Gorenberg (12329728)

    Published 2022
    “…A total of 15 palmitoylated proteins are significantly differentially expressed in whole brain (3 decreased, including PPT1; 12 increased; 1.5-fold, <i>p</i> < 0.05; blue lines). …”
  8. 11948
  9. 11949
  10. 11950
  11. 11951
  12. 11952

    The RBPJ AOS variants show dysregulated Notch target gene expression when compared to WT RBPJ. by Ellen K. Gagliani (11478756)

    Published 2022
    “…Cells were additionally transfected with increasing concentrations (50, 150 & 250 ng) of either WT, NBM (F261A/V263A/R422E/E425R/E398R), KRS (K195E/R218H/S221D), NBM/KRS, or K195E Rbpj constructs in panels <b><i>ii-iv</i></b>. …”
  13. 11953

    Image_1_MiR-185-5p Protects Against Angiogenesis in Polycystic Ovary Syndrome by Targeting VEGFA.tif by Jingzan Wei (9121817)

    Published 2020
    “…Although miR-185-5p (miR-185) was decreased in PCOS patients, the exact function of miR-185 on PCOS development still requires further investigation. …”
  14. 11954
  15. 11955
  16. 11956
  17. 11957
  18. 11958
  19. 11959
  20. 11960