Photocatalytic CO Oxidation over Nanoparticulate Au-Modified TiO₂ Aerogels: The Importance of Size and Intimacy by Ashley M. Pennington (4888408)

“…We determine the activity of catalytic carbon monoxide (CO) oxidation as a function of the size of gold nanoparticles (Au NP), their weight loading, and their interfacial intimacy with similarly sized TiO₂ NPs covalently bonded in an aerogel support. …”

Photocatalytic CO Oxidation over Nanoparticulate Au-Modified TiO₂ Aerogels: The Importance of Size and Intimacy by Ashley M. Pennington (4888408)

“…We determine the activity of catalytic carbon monoxide (CO) oxidation as a function of the size of gold nanoparticles (Au NP), their weight loading, and their interfacial intimacy with similarly sized TiO₂ NPs covalently bonded in an aerogel support. …”

Photocatalytic CO Oxidation over Nanoparticulate Au-Modified TiO₂ Aerogels: The Importance of Size and Intimacy by Ashley M. Pennington (4888408)

“…We determine the activity of catalytic carbon monoxide (CO) oxidation as a function of the size of gold nanoparticles (Au NP), their weight loading, and their interfacial intimacy with similarly sized TiO₂ NPs covalently bonded in an aerogel support. …”

Photocatalytic CO Oxidation over Nanoparticulate Au-Modified TiO₂ Aerogels: The Importance of Size and Intimacy by Ashley M. Pennington (4888408)

“…We determine the activity of catalytic carbon monoxide (CO) oxidation as a function of the size of gold nanoparticles (Au NP), their weight loading, and their interfacial intimacy with similarly sized TiO₂ NPs covalently bonded in an aerogel support. …”

Photocatalytic CO Oxidation over Nanoparticulate Au-Modified TiO₂ Aerogels: The Importance of Size and Intimacy by Ashley M. Pennington (4888408)

“…We determine the activity of catalytic carbon monoxide (CO) oxidation as a function of the size of gold nanoparticles (Au NP), their weight loading, and their interfacial intimacy with similarly sized TiO₂ NPs covalently bonded in an aerogel support. …”

CXCR4 was upregulated on less differentiated T cell subsets during sepsis. by Kimberly M. Ramonell (4678906)

The size of particle free areas (holes) characterize the simulated patterning process. by Előd Méhes (184347)

“…B: Expansion rate of individual holes, as a function of their size. We identified areas that did not merge with adjacent holes during a 30 minute time interval, and determined the change in their size. …”

Log2 fold changes in ATRX knockdown HFFs compared to control HFFs. by Anne-Charlotte Stilp (3520415)

Photomicrographs showing NFILS-IR nerve fibers in TPL cervix (a) and a NFILS-IR nerve fascicle in NP corpus (b) by Berith K Tingåker (87814)

“…There is a significant decrease of IR-nerve fibres/mmin the term pregnant group compared to non-pregnant controls, p < 0.0001. …”

Expression of HIF-1α in qPCR and Western blot analysis at different post-mortem times. by Salomé Mascarell (20234689)

A possible involvement of the IκBζ pathway in IL-17A-induced hypercontractility. by Hirotada Akiho (559455)

“…Scale bars, 50 µm. (<b>D</b>) Immunofluorescence staining of NFκB p65 protein in primary cultured murine SMCs after 30 min treatment with IL-17A or IL-1β. …”

Fig 5 - by Debarpan Dhar (724473)

<i>Leishmania</i> parasites upregulate c-MET mRNA expression in neutrophils. by Katiuska Passelli (11965496)

GQ-16 treatment increases UCP-1 expression in interscapular brown adipose tissue. by Michella S. Coelho (2637202)

GQ-16 treatment induces UCP-1 expression in subcutaneous white adipose tissue. by Michella S. Coelho (2637202)

GQ-16 treatment reduces hepatic triglyceride accumulation. by Michella S. Coelho (2637202)

GQ-16 treatment induces UCP-1 expression in visceral white adipose tissue. by Michella S. Coelho (2637202)

Phosphatidylserine activates the BDNF-mediated signaling pathway. by Sivan Yannai (3594626)

Mcl-1 degradation is slowed by a T163E mutation (but not T163A) in transfected CHO cells. by Shanna K. Nifoussi (128297)

“…In Panel B, the decline in Mcl-1 expression at 3 hours in 2 independent experiments ranged from 53–66% with WT-Mcl-1, 25–54% with Mcl-1-S162A, and 25–50% with Mcl-1-T163A. While the decline in expression with WT-Mcl-1 appeared be slightly greater than that seen with Mcl-1-T163A, this was not a consistent finding. …”

Epithelial cell death induced by cFlip deletion is counteracted by vFLIP <i>in vivo</i>. by Barbara Ruder (8379354)