DataSheet1_Defining the altered glycoproteomic space of the early secretory pathway by class I mannosidase pharmacological inhibition.pdf by Cristian V. A. Munteanu (8610765)

“…<p>N-glycosylation is a key process for various biological functions like protein folding, maturation and sorting for the conventional secretory compartment, cell-cell communication and immune response. …”

Table1_Defining the altered glycoproteomic space of the early secretory pathway by class I mannosidase pharmacological inhibition.XLSX by Cristian V. A. Munteanu (8610765)

“…<p>N-glycosylation is a key process for various biological functions like protein folding, maturation and sorting for the conventional secretory compartment, cell-cell communication and immune response. …”

Aortic and Mitral Valve Replacement With Reconstruction of the Intervalvular Fibrous Body: How to Do the Commando Procedure by Carolina Barajas Díaz (8107588)

“…</p><p>This technique is a complex procedure with a high operative morbidity and mortality, but one must not forget that in some cases, it is the only option available to treat these patients (2,4,7). Thus, a framework of technical considerations can help surgical teams achieve reproducible success with this technique, decreasing the technical failures (3). …”

video3_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.mp4 by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

Image1_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.tiff by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

video3_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.mp4 by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

video1_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.mp4 by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

Dataset. by Naved Alam (17227106)

“…More than half of the factory workers (53.2%) were tobacco users. Tobacco users were 3.52 times more likely to have a severe DMFT index. …”

video1_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.mp4 by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

Image1_Neospora caninum Infection Triggers S-phase Arrest and Alters Nuclear Characteristics in Primary Bovine Endothelial Host Cells.tiff by Zahady D. Velásquez (6724274)

“…Analyses on key molecules of cell cycle regulation documented a significant alteration of cyclin A2 and cyclin B1 abundance in N. caninum-infected host endothelial cells, thereby confirming irregularities in the S-phase and S-to-G2/M-phase transition. …”

Protein disulfide isomerases (PDIs) negatively regulate ebolavirus structural glycoprotein expression in the endoplasmic reticulum (ER) via the autophagy-lysosomal pathway by Bin Wang (30851)

“…We suggest that ebolaviruses hijack the host protein folding and ERAD machinery to increase their fitness via reticulophagy during infection.…”

Acoustic parameters of voices of women in post-menopause by Renata D'Arc Scarpel (12818055)

“…METHOD: Quantitative cross-sectional study was conducted with 23 women in the postmenopausal period, using the Praat software 5.2.0. The parameters considered were fundamental frequency, jitter, shimmer, harmonic noise and spectrographic analysis. …”

Inhibiting autophagy reduces retinal degeneration caused by protein misfolding by Jingyu Yao (322837)

Evaluation of plasma assisted noninvasive surgery (PANIS) as a new approach for the treatment of conjunctivochalasis; a clinical case series by Khosrow Jadidi (3376994)

“…In all patients, the grade 3 and 4 CCh were decreased to grade 1 and 2, respectively, after one month all treated eyes were normal and no interface complications were observed.…”

Data_Sheet_1_Antivirulence activities of retinoic acids against Staphylococcus aureus.docx by Inji Park (16975800)

“…Furthermore, transcriptional analysis disclosed that 13-cis-retinoic acid repressed the expressions of virulence- and biofilm-related genes, such as the two-component arlRS system, α-hemolysin hla, nuclease (nuc1 and nuc2), and psmα (phenol soluble modulins α) in S. aureus. …”

Table 1_Pan-cancer multi-omics analysis of CCT4 in tumor progression and cancer immunity, with focus on lung adenocarcinoma.docx by Yonghui Wu (171826)

“…High CCT4 levels were linked to reduced immune cell infiltration and diminished anti-tumor immune signaling, specifically manifested as increased Th2 cell infiltration and decreased Th1 and CD8⁺ T-cell signatures. …”

Table_1_Refractory Helicobacter pylori infection and the gastric microbiota.docx by Dongsheng Liu (73360)

“…The bacterial interaction network diagram suggested that the microbiota interactions in the refractory H. pylori infection group decreased. The gastric microbiota of the refractory H. pylori infection group was enriched in the pathways of metabolism and infectious diseases (energy metabolism, bacterial secretion system, glutathione metabolism, protein folding and associated processing, sulphur metabolism, membrane and intracellular structural molecules, lipopolysaccharide biosynthesis, ubiquinone and other terpenoid-quinone biosynthesis, inorganic ion transport and metabolism, and metabolism of cofactors and vitamins) when compared with the H. pylori-positive group without treatment based on PICRUSt analysis.…”

Image_1_Refractory Helicobacter pylori infection and the gastric microbiota.pdf by Dongsheng Liu (73360)

“…The bacterial interaction network diagram suggested that the microbiota interactions in the refractory H. pylori infection group decreased. The gastric microbiota of the refractory H. pylori infection group was enriched in the pathways of metabolism and infectious diseases (energy metabolism, bacterial secretion system, glutathione metabolism, protein folding and associated processing, sulphur metabolism, membrane and intracellular structural molecules, lipopolysaccharide biosynthesis, ubiquinone and other terpenoid-quinone biosynthesis, inorganic ion transport and metabolism, and metabolism of cofactors and vitamins) when compared with the H. pylori-positive group without treatment based on PICRUSt analysis.…”

Data Sheet 1_Pan-cancer multi-omics analysis of CCT4 in tumor progression and cancer immunity, with focus on lung adenocarcinoma.docx by Yonghui Wu (171826)

“…High CCT4 levels were linked to reduced immune cell infiltration and diminished anti-tumor immune signaling, specifically manifested as increased Th2 cell infiltration and decreased Th1 and CD8⁺ T-cell signatures. …”

DataSheet_1_Refractory Helicobacter pylori infection and the gastric microbiota.pdf by Dongsheng Liu (73360)

“…The bacterial interaction network diagram suggested that the microbiota interactions in the refractory H. pylori infection group decreased. The gastric microbiota of the refractory H. pylori infection group was enriched in the pathways of metabolism and infectious diseases (energy metabolism, bacterial secretion system, glutathione metabolism, protein folding and associated processing, sulphur metabolism, membrane and intracellular structural molecules, lipopolysaccharide biosynthesis, ubiquinone and other terpenoid-quinone biosynthesis, inorganic ion transport and metabolism, and metabolism of cofactors and vitamins) when compared with the H. pylori-positive group without treatment based on PICRUSt analysis.…”