LIN-2 and FRM-3 regulate the synaptic abundance but not surface expression level of AChRs. by Lei Li (29537)

“…Representative traces (E, G) and mean current amplitude (F, H) are shown. (I, J) GABA-activated currents were decreased by 50% in <i>lin-2</i>^<i>null</i> mutants but were unchanged in <i>frm-3</i>^<i>null</i> mutants. …”

Recognition of <i>F. novicida</i> strains by lysophagosomes and lysosomes in HeLa–FcγRII cells. by Dhandy Koesoemo Wardhana (21956781)

Inhibition experiments with PP PLA₂. by Catherine A. Vulfius (676708)

Recognition of <i>F. novicida</i> strains by the mitochondria and Golgi complex in HeLa–FcγRII cells. by Dhandy Koesoemo Wardhana (21956781)

DataSheet1_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

DataSheet2_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

Overexpression of miR-184 disrupts the blood-brain barrier and larval locomotion. by Sravya Paluri (5475260)

SDS-PAGE analysis of enzymes. by Lina Liu (227490)

Compromised angiogenesis and vascular Integrity in impaired diabetic wound healing by Uzoagu A. Okonkwo (8754933)

“…Specifically, the expression of VEGF-A, Sprouty2, PEDF, LRP6, Thrombospondin 1, CXCL10, CXCR3, PDGFR-β, HB-EGF, EGFR, TGF-β1, Semaphorin3a, Neuropilin 1, angiopoietin 2, NG2, and RGS5 were down-regulated in diabetic wounds. …”

Post-response beta activity. by Wen Wen (596162)

Intracellular growth of the wild-type strain and <i>FTN_0096</i> mutant in THP-1 cells. by Dhandy Koesoemo Wardhana (21956781)

Loss of expression of <i>dbt</i> in <i>tim-</i>GAL4-expressing neurons can lead to a sleep decrease during the nighttime, while loss of expression of <i>dbt</i> with <i>per-</i>GAL... by Jialin Wang (490082)

Representative images of the right eye of a 51-year-old woman with active central serous chorioretinopathy. by Dae Joong Ma (5687885)

Intracellular growth and cytotoxicity of the wild-type strain and <i>FTN_0096</i> mutant in HeLa–FcγRII cells. by Dhandy Koesoemo Wardhana (21956781)

Fig 2 - by Diana Xochiquetzal Robledo-Cadena (5265959)

Virus evolution alters the entry kinetics of autologous CAP256 viruses. by Lucia Reh (546554)

Effects of 50 µM stearic acid, oleic acid and linoleic acid on cell death and apoptosis of 3T3L1 preadipocytes. by Ming-Che Shen (629982)

Loss of exostosin-1 (EXT1), specific reduction of heparan sulfate (HS) in fibroblasts, and decreased acidic mucosubstances in the stroma of <i>S100a4-Cre; Ext1</i><sup><i>f/f</i></... by Ayumi Niwa (6256790)

image2_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”