Initial treatment regimen. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

Patients baseline characteristics. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

Overall survival. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

Progression-free survival by treatment. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

Maintenance regimen and adverse event management. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

BCC-specific survival. by Shivani Patel (4742748)

“…Hedgehog inhibition (HHI) with vismodegib or sonidegib induces a 50–60% response rate. Long-term toxicity includes muscle spasms and weight loss leading to dose decreases. …”

Primer sequences and sizes. by Peijun Liu (563547)

<i>USP28</i> protein level was decreased by hypoxia signaling via both protein degradation and transcriptional repression in A549 cells. by Qin Li (36669)

“…(C) Hypoxia decreased <i>USP28</i> mRNA levels in A549 cells. A549 cells were cultured in 1% O₂ for 24 hr. …”

DAF-16/FOXO is Required for Locomotory Healthspan and Lifespan Benefits of Genetic Glycolysis Disruption. by Brian Onken (86790)

“…<i>daf-16</i> mutants are documented to have lowered locomotory ability vs. WT later in life [<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1008982#pgen.1008982.ref091" target="_blank">91</a>, <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1008982#pgen.1008982.ref092" target="_blank">92</a>]; we note that, although comparison of scores across experiments is not possible due to variation in baseline values, the swimming rate of <i>daf-16(mgDf50)</i> control animals was slightly lower than WT controls on the same day of life (in <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1008982#pgen.1008982.g001" target="_blank">Fig 1C</a>; mean of 46.17 head thrashes/30 sec. for <i>daf-16</i> vs. 52.17 head thrashes/30 sec. for WT on day 10). …”

Attenuation of Quorum Sensing Regulated Virulence of <i>Pectobacterium carotovorum</i> subsp. <i>carotovorum</i> through an AHL Lactonase Produced by <i>Lysinibacillus</i> sp. Gs50 by Sneha S. Garge (3375242)

“…Our results demonstrate the potential of <i>Lysinibacillus</i> sp. Gs50 as a preventive and curative biocontrol agent.…”

Effect of AGPAT3 downregulation on the mTORC1 signaling pathway. by Hadi Alizadeh (20854757)

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Introduction of potential genes and signal pathways related to cisplatin resistance in the A2780 cell line based on Bioinformatics analysis. by Hadi Alizadeh (20854757)

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Mechanism of AGPAT3-mediated mTORC1 activation and its downstream effects. by Hadi Alizadeh (20854757)

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Effect of AGPAT3 overexpression on mTORC1 signaling pathway. by Hadi Alizadeh (20854757)

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Differentially expressed genes in A2780 resistant and sensitive to cisplatin ovarian cancer originated cell lines. by Hadi Alizadeh (20854757)

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Cell cycle analysis of A2780 cells following AGPAT3 overexpression. by Hadi Alizadeh (20854757)

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Characterization of cisplatin resistance phenotype in A2780 and A2780cp cells. by Hadi Alizadeh (20854757)

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Viability and drug resistance of A2780 cells following AGPAT3 overexpression. by Hadi Alizadeh (20854757)

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Presenting the original images of Western blotting analysis. by Hadi Alizadeh (20854757)

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Analysis of MD simulations for the SP domain of native PC and its mutant structures (L305R, W342C, G403R, V420E, and W444C) over 200 ns simulations. by Mahvash Farajzadeh-Dehkordi (17468232)