Table_1_Indicators of Health-Related Quality of Life in Cats With Degenerative Joint Disease: Systematic Review and Proposal of a Conceptual Framework.docx by Gillian Yeowell (11720489)

Video_1_Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome.MP4 by Lee J. Martin (9658109)

“…It is uncertain whether this degeneration of MNs is triggered intrinsically and is autonomous, or if the disease initiating mechanisms are extrinsic to MNs. We hypothesized that skeletal muscle is a primary site of pathogenesis in ALS that triggers MN degeneration. …”

The lag phase defect is specific to transitions that require the production of FA, and correlates with FA concentration. by Zachary T. Hying (21512605)

Concentrations of C₁ metabolites are imbalanced in strains that cannot maintain FA homeostasis during the transition from formate to MeOH. by Zachary T. Hying (21512605)

ExaF contributes to FA homeostasis by lowering the FA concentration. by Zachary T. Hying (21512605)

<i>efgA</i> and <i>ttmR</i> affect the transition to methylotrophy independently. by Zachary T. Hying (21512605)

Relative metabolic cost of muscle function groups across speeds in the simulation workflows. by Israel Luis (13916706)

DataSheet1_The efficacy and safety of dachaihu decoction in the treatment of type 2 diabetes mellitus: A systematic review and meta-analysis.docx by Zehua Zhang (312656)

“…<p>Background: Type 2 diabetes mellitus (T2DM) is a clinical metabolic syndrome characterized by persistent hyperglycemia, which is caused by defective insulin secretion and decreased function in regulating glucose metabolism. …”

SD11 modulates gut integrity via the upregulation of tight junction proteins in the colon of STZ-induced diabetic mice. by Waraporn Ruathong (20308203)

SD11 downregulates colonic inflammatory responses in STZ-induced diabetic mice. by Waraporn Ruathong (20308203)

Image_2_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_1_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_3_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_8_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_9_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_4_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_7_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_5_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_6_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)

Image_10_Stimulation of Let-7 Maturation by Metformin Improved the Response to Tyrosine Kinase Inhibitor Therapy in an m6A Dependent Manner.tif by Kai Li (205203)