Supplementary file 1_Water stress reduces cellulose deposition in the cell wall and increases wax content, resulting in decreased fiber quality.docx by Yongchao Han (10797698)

“…At 30 DPA, compared to the WW treatment, the sucrose contents in SD217 and XC7 fibers decreased by 18.66% and 12.85%, while cellulose contents dropped by 9.91% and 17.17%, respectively, resulting in a significant decrease in the thickness of the cell walls by 10.59% and 9.50% respectively. …”

Supplementary file 2_Water stress reduces cellulose deposition in the cell wall and increases wax content, resulting in decreased fiber quality.xlsx by Yongchao Han (10797698)

“…At 30 DPA, compared to the WW treatment, the sucrose contents in SD217 and XC7 fibers decreased by 18.66% and 12.85%, while cellulose contents dropped by 9.91% and 17.17%, respectively, resulting in a significant decrease in the thickness of the cell walls by 10.59% and 9.50% respectively. …”

<i>sirB⁻</i>, Vic1, Vic2 and T6SS-double mutants showed decreased virulence in planta. by Johanna Nykyri (124297)

Increased Dietary Intake of Saturated Fatty Acid Heptadecanoic Acid (C17:0) Associated with Decreasing Ferritin and Alleviated Metabolic Syndrome in Dolphins by Stephanie K. Venn-Watson (774138)

“…To assess potential protective factors against metabolic syndrome related to fish diets, fatty acids were compared between two dolphin populations with higher (n = 30, Group A) and lower (n = 19, Group B) mean insulin (11 ± 12 and 2 ± 5 μIU/ml, respectively; <i>P</i> < 0.0001) and their dietary fish. …”

Fig 2 - by Kyu Jin Lee (18143946)

“…<p>In-hospital mortality rates across the quartiles for SI, DSI, MSI, and Age-SI values among patients with normal LVEF (≥ 50%; A, B, C, and D, respectively) and those with decreased LVEF (< 50%; E, F, G, and H, respectively). …”

Fig 4 - by Kyu Jin Lee (18143946)

Fig 1 - by Ilari Kuitunen (11084096)

Fig 2 - by Ilari Kuitunen (11084096)

Image_4_Repeated Inoculation of Young Calves With Rumen Microbiota Does Not Significantly Modulate the Rumen Prokaryotic Microbiota Consistently but Decreases Diarrhea.TIF by Dengpan Bu (369549)

Image_1_Repeated Inoculation of Young Calves With Rumen Microbiota Does Not Significantly Modulate the Rumen Prokaryotic Microbiota Consistently but Decreases Diarrhea.TIF by Dengpan Bu (369549)

Image_3_Repeated Inoculation of Young Calves With Rumen Microbiota Does Not Significantly Modulate the Rumen Prokaryotic Microbiota Consistently but Decreases Diarrhea.TIF by Dengpan Bu (369549)

Image_5_Repeated Inoculation of Young Calves With Rumen Microbiota Does Not Significantly Modulate the Rumen Prokaryotic Microbiota Consistently but Decreases Diarrhea.TIF by Dengpan Bu (369549)

Image_2_Repeated Inoculation of Young Calves With Rumen Microbiota Does Not Significantly Modulate the Rumen Prokaryotic Microbiota Consistently but Decreases Diarrhea.TIF by Dengpan Bu (369549)

β₂-Agonist Induced cAMP Is Decreased in Asthmatic Airway Smooth Muscle Due to Increased PDE4D by Thomas Trian (221535)

“…</p> <h3>Results</h3><p>In the presence of IBMX, a pan PDE inhibitor, asthmatic ASM had ∼50% lower cAMP production in response to isoproterenol, albuterol, formoterol, and forskolin compared to non-asthmatic ASM. …”

Flow chart for the enrolled patients. by Kyu Jin Lee (18143946)

“…Left ventricular (LV) function was categorized as either normal LV ejection fraction (LVEF ≥ 50%) or decreased LVEF (<50%).</p><p>Results</p><p>Among the 1,194 patients with septic shock, 392 (32.8%) who underwent echocardiography within 24 h of time zero were included in the final analysis (normal LVEF: n = 246; decreased LVEF: n = 146). …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image3_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”