Loss of expression of <i>dbt</i> in <i>tim-</i>GAL4-expressing neurons can lead to a sleep decrease during the nighttime, while loss of expression of <i>dbt</i> with <i>per-</i>GAL... by Jialin Wang (490082)

Representative images of the right eye of a 51-year-old woman with active central serous chorioretinopathy. by Dae Joong Ma (5687885)

Intracellular growth and cytotoxicity of the wild-type strain and <i>FTN_0096</i> mutant in HeLa–FcγRII cells. by Dhandy Koesoemo Wardhana (21956781)

Fig 2 - by Diana Xochiquetzal Robledo-Cadena (5265959)

Virus evolution alters the entry kinetics of autologous CAP256 viruses. by Lucia Reh (546554)

Effects of 50 µM stearic acid, oleic acid and linoleic acid on cell death and apoptosis of 3T3L1 preadipocytes. by Ming-Che Shen (629982)

Loss of exostosin-1 (EXT1), specific reduction of heparan sulfate (HS) in fibroblasts, and decreased acidic mucosubstances in the stroma of <i>S100a4-Cre; Ext1</i><sup><i>f/f</i></... by Ayumi Niwa (6256790)

Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

“…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”

Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

“…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”

Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

“…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”

Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

“…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”

Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

“…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”

image2_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image3_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

presentation1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.pptx by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image1_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”

image3_Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways.tif by Xin Zhi (1829224)

“…Specifically, myostatin increased the phosphorylation of Smad2, which led to the activation of NF-κB and MAPK pathways to activate osteoclastogenesis. Ccdc50 was identified as a gene whose expression was highly decreased in osteoclastogenesis upon myostatin treatment, and it could inhibit the function of myostatin in osteoclastogenesis by blocking NF-κB and MAPKs pathways. …”