Identification of Two Plastid Proteins in the Dinoflagellate <i>Alexandrium affine</i> That Are Substantially Down-Regulated by Nitrogen-Depletion by Fred Wang-Fat Lee (2339839)

“…Five of these nine spots were all identified as isoforms of the plastid Form II ribulose-1,5 bisphosphate carboxylase/oxygenase (Rubisco II), while an additional four protein spots with a molecular weight of 50 kDa were identified as isoforms of a novel protein named nitrogen-associated protein 50 (NAP50). …”

Identification of Two Plastid Proteins in the Dinoflagellate <i>Alexandrium affine</i> That Are Substantially Down-Regulated by Nitrogen-Depletion by Fred Wang-Fat Lee (2339839)

“…Five of these nine spots were all identified as isoforms of the plastid Form II ribulose-1,5 bisphosphate carboxylase/oxygenase (Rubisco II), while an additional four protein spots with a molecular weight of 50 kDa were identified as isoforms of a novel protein named nitrogen-associated protein 50 (NAP50). …”

Identification of Two Plastid Proteins in the Dinoflagellate <i>Alexandrium affine</i> That Are Substantially Down-Regulated by Nitrogen-Depletion by Fred Wang-Fat Lee (2339839)

“…Five of these nine spots were all identified as isoforms of the plastid Form II ribulose-1,5 bisphosphate carboxylase/oxygenase (Rubisco II), while an additional four protein spots with a molecular weight of 50 kDa were identified as isoforms of a novel protein named nitrogen-associated protein 50 (NAP50). …”

Mean absorbance levels of lytic activity in parasitized and non-parasitized nymphs and adults of the whitefly <i>T</i>. <i>vaporariorum</i>. by Jorge Contreras-Garduño (3275727)

Syndecan-1 expression along the basolateral endothelial surface does not change in inflamed venules. by Paulina M. Kowalewska (625078)

AZP-531, an unacylated ghrelin analog, improves food-related behavior in patients with Prader-Willi syndrome: A randomized placebo-controlled trial by Soraya Allas (4752756)

“…AZP-531, a first-in-class UAG analog, was shown to inhibit the orexigenic effect of AG in animals, to improve glycemic control and decrease body weight in humans. …”

Effects of feed additives on alpha diversity. by Yoshiaki Sato (2170141)

Cartilaginous extracellular matrix space was severely reduced in <i>Slc35a3</i>^{<i>–/–</i>}mice. by Soichiro Saito (15285875)

Syndecan-1 expression in the abdominal wall and plasma does not change after LTA injection. by Paulina M. Kowalewska (625078)

Essential oil-incorporated carbon nanotubes filters for bacterial removal and inactivation - Fig 4 by Xiuli Dong (658906)

Hand choice. by Aoife M. Fitzpatrick (13955365)

Essential oil-incorporated carbon nanotubes filters for bacterial removal and inactivation - Fig 2 by Xiuli Dong (658906)

The percentage of NaCl tolerant <i>E</i>. <i>coli</i> cells retained on filters. by Xiuli Dong (658906)

<i>Becn1</i>^F121A leads to autophagy hyperactivation in vivo. by Altea Rocchi (3278457)

<i>Sdc1</i>^-/- mice have normal leukocyte recruitment to the parietal peritoneum microcirculation. by Paulina M. Kowalewska (625078)

“…<p>Wild-type and <i>Sdc1</i>^-/- mice were injected IP with 50<i> µ</i>L of saline, <i>S. aureus</i> LTA (125<i> µ</i>g), <i>E. coli</i> LPS (125<i> µ</i>g) or TNF<i>α</i> (500 ng). …”

NSAIDs decrease the viability of ovarian cancer cells in a dose-dependent manner. by Blanca L. Valle (406582)

Map of 91 federal, state, and local parks and protected area (PPA) study sites overlaid on Level 2 ecoregion. by Kira Minehart (21029571)

T198A/G substitution markedly decrease p27 protein stability. by Monica Schiappacassi (227672)

Decrease in viability in response to KDM6A/p21^CIP1 depletion in E7 expressing cells may be a consequence of replication stress. by David R. Soto (4484626)

The effects of pretreatment with fingolimod-phosphate on BMECs in the presence of MS sera. by Hideaki Nishihara (545149)

“…Pretreatment with fingolimod-phosphate resulted in an increase in the claudin-5 protein levels (B) and TEER values (G) and a decrease in the VCAM-1 protein levels (F) in the BMECs, which were upregulated after exposure to the sera from all MS patients, including the RRMS-R, RRMS-S and SPMS patients, compared to that observed in the cells not pretreated with fingolimod-phosphate.…”