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we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
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a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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13981
DataSheet3_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13982
Table1_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.docx
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13983
DataSheet4_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13984
Table2_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.docx
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13985
DataSheet6_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13986
DataSheet2_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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13987
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13988
Sodium butyrate alleviates potential Alzheimer’s disease in vitro by suppressing Aβ and tau activation and ameliorates Aβ-induced toxicity
Published 2023“…Sodium butyrate (NaB) is an HDAC inhibitor profoundly found to be neuroprotective. We have investigated the neuroprotective effects of NaB in SH-SY5Y cells stimulated with TNF-α/Aβ in SH-SY5Y cells and LPS-induced BV-2 cells. …”
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13989
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13990
Table4_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13991
Table2_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13992
Table3_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13993
Image6_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13994
Table1_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13995
Image1_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13996
Image4_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13997
Image2_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13998
Image3_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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13999
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14000
Unveiling the Cytotoxic Potential of Quercetin-Loaded Magnetic Bacterial Bots against Cervical Cancer
Published 2025“…The cytotoxicity of the fabricated magnetic bacterial bots within the low dose (5 × 10<sup>5</sup> CFU/mL) against the HeLa monolayer and 3D spheroid model was found around 84%, which had remarkably enhanced up to more than 91% in the presence of an external magnet. …”