mtDNA mutator flies exhibit shortened lifespan, a locomotion defect, neuron loss, and mitochondrial dysfunction. by Colby L. Samstag (5987945)

“…<b>(C)</b> Quantification of dopaminergic neurons within the indicated clusters of in 50-day-old control and 2xPolG^mut flies. …”

Is prehospital endobronchial intubation a risk factor for subsequent ventilator associated pneumonia? A retrospective analysis by Ana Catalina Hernandez Padilla (6730013)

Comprehensive prediction process of shape errors. by Zhe Hu (787283)

Data_Sheet_1_Rho-Kinase Inhibition Ameliorates Dasatinib-Induced Endothelial Dysfunction and Pulmonary Hypertension.docx by Csilla Fazakas (218764)

IGF2 knockdown alters the cellular response to Taxol. by Jurriaan Brouwer-Visser (578686)

“…**p<0.01; One-way ANOVA with Bonferroni posttest. The bars show the mean±SEM of two independent experiments.</p>…”

Image_3_Rho-Kinase Inhibition Ameliorates Dasatinib-Induced Endothelial Dysfunction and Pulmonary Hypertension.tif by Csilla Fazakas (218764)

Image_2_Rho-Kinase Inhibition Ameliorates Dasatinib-Induced Endothelial Dysfunction and Pulmonary Hypertension.tif by Csilla Fazakas (218764)

The effect of VEGFR-2 shRNA on tumorigenesis, angiogenesis, self-renewal and VM formation by GSLCs. by Xiaohong Yao (383008)

“…Nuclei were stained with DAPI (blue). Scale bar = 50 µm. * Indicates significantly decreased number of CD133-positive cells in mice bearing tumors formed by VEGFR-2 knock-down GSLCs (<i>p<0.05</i>). …”

Inverse relationship between the presence of Bax and Rad51. by Loro L. Kujjo (354585)

“…Values for each sample were determined as mean ± SEM per oocyte (AKR/J n = 50; C57BL/6 n = 50), and comparison was made between the two strains using Student's t-test. …”

Combination therapy of high dose PC1, kansui, and JQ1 reactivates latent HIV in a primary CD4+ T cell model of HIV latency. by Daniele C. Cary (3559394)

“…At 12 days post infection, cells were stimulated for 24 hours with: (A) PC1 (24 μM) and kansui (50 μg/ml) (B) PC1 (24 μM) and JQ1 (0.1 μM) (C) e PC1 (24 μM), kansui (50 μg/ml), and JQ1 (0.1 μM). …”

Combination therapy of low dose PC1, kansui, and JQ1 reactivates latent HIV in a primary CD4+ T cell model of HIV latency. by Daniele C. Cary (3559394)

“…At 12 days post infection, cells were stimulated for 24 hours with: (A) single and double PC1 (6 μM) and kansui (50 μg/ml) (B) single and double PC1 (6 μM) and JQ1 (0.1 μM) (C) single and triple PC1 (6 μM), kansui (50 μg/ml), and JQ1 (0.1 μM). …”

Synthesis, Structure, and Magnetic Properties of Dy₂Co₂<i>L</i>₁₀(bipy)₂ and <i>Ln</i>₂Ni₂<i>L</i>₁₀(bi... by Fang-Hua Zhao (2038318)

“…The 3,5-dichlorobenzoate anion, <i>L</i>^–, serves as a bridging ligand and 2,2′-bipyridine, bipy, as a terminal bidentate ligand to yield, through hydrothermal syntheses, the tetranuclear clusters Dy₂Co₂<i>L</i>₁₀(bipy)₂, <b>1</b>, and <i>Ln</i>₂Ni₂<i>L</i>₁₀(bipy)₂, where <i>Ln</i> is the trivalent La, <b>2</b>, Gd, <b>3</b>, Tb, <b>4</b>, Dy, <b>5</b>, or Ho, <b>6</b>, ion. …”

Subgroup analysis. by Bruno Martins Tomazini (6509528)

“…<div><p>Background</p><p>Halofuginone (PJS-539) is an oral prolyl-tRNA synthetase inhibitor that has a potent <i>in vitro</i> activity against SARS-CoV-2 virus. …”

Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival by Meng Li (79487)

“…<p>Knockdown SIRT3 caused ATF4 protein decrease via translation regulation. <b>A,</b> Western blots show ATF4 protein levels in different DLBCL cells with control or SIRT3 shRNAs. …”

Study outcomes. by Bruno Martins Tomazini (6509528)

“…<div><p>Background</p><p>Halofuginone (PJS-539) is an oral prolyl-tRNA synthetase inhibitor that has a potent <i>in vitro</i> activity against SARS-CoV-2 virus. …”

Image 6_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 3_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xlsx by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 3_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 7_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 2_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”