Showing 14,381 - 14,400 results of 106,497 for search '(( 50 ((mean decrease) OR (we decrease)) ) OR ( 5 ((teer decrease) OR (a decrease)) ))', query time: 1.70s Refine Results
  1. 14381
  2. 14382
  3. 14383
  4. 14384
  5. 14385
  6. 14386

    Table_5_Effectiveness and tolerability of different therapies in preventive treatment of MOG-IgG-associated disorder: A network meta-analysis.docx by Xiaofei Wang (135107)

    Published 2022
    “…AZA, MMF, OC and RTX showed a trend to decrease ARR, but those results did not reach significant differences. …”
  7. 14387

    PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  8. 14388

    PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  9. 14389

    PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  10. 14390

    PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  11. 14391

    PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  12. 14392
  13. 14393
  14. 14394
  15. 14395

    Impact of TLR5 knockout on specific taxa in the fecal microbiota of mice and differences in comparing two colonies (TLR5KO1 and TLR5KO2) bred in different laboratories. by Wei Zhang (405)

    Published 2016
    “…<p>Heat map showing statistically significant changes (p<0.05) on different fecal taxa. Red shows a significant increase, blue signifies a significant decrease, and black shows not found. …”
  16. 14396
  17. 14397

    Juvenile demyelination impairs the maturation of PV interneurons in the PFC. by Sara Hijazi (21656615)

    Published 2025
    “…Representative traces of voltage responses following +450 pA current injection. Scale bar: 100 ms, 10 mV. …”
  18. 14398

    Image1_RETRACTED: miR-16 and Fluoxetine Both Reverse Autophagic and Apoptotic Change in Chronic Unpredictable Mild Stress Model Rats.tif by Yang Yang (45629)

    Published 2025
    “…Expression levels of miR-16 and BDNF in the hippocampus were examined with RT-PCR, and it was found that increased 5-HT2a receptor expression induced by CUMS can be decreased by miR-16 and Fluoxetine administration. …”
  19. 14399

    All data points from Fig 2. by Sara Hijazi (21656615)

    Published 2025
    “…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
  20. 14400

    All data points from Fig 8. by Sara Hijazi (21656615)

    Published 2025
    “…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”