ER stress sensors and EBI3 are expressed during chondrogenic differentiation of MSCs. by Tong Zhang (103827)

Fig 5 - by Debarpan Dhar (724473)

LZP is required for TG secretion. by Jiao-Xiang Wu (6323045)

Astrocytic expression of Pla2g3 by chronic oxidative stress. by Daishi Yui (795083)

GQ-16 reduces brown adipose tissue mass and adipocyte diameter. by Michella S. Coelho (2637202)

Increased expression of Pla2g3 in frontal cortex of AD patients. by Daishi Yui (795083)

Reduced IRF3 and TBK1 phosphorylation in ATRX knockdown HFFs. by Anne-Charlotte Stilp (3520415)

Analgesic effect of a cholinergic agonist (carbachol) in a sural nerve ligation-induced hypersensitivity mouse model by Nodoka Kato (18298748)

“…</p> <p>SN ligation resulted in a significant decrease in pain threshold for mechanical stimulation 1 day after ligation. …”

DICER+AGO2 enzymes cleave fluorogenic substrates into duplex products that are unstable at T>25°C. by Mark A. Bernard (608655)

Minocycline administration reduced CCR2-positive cells in RPE flat-mount. by Ryo Terauchi (10687481)

The population of Ly6C positive circulating monocytes in minocycline- and control-treated <i>Mertk</i>^{<i>−/−</i>}<i>Cx3cr1</i>^<i>GFP/+</i><i>Ccr2</i><sup><i>RF... by Ryo Terauchi (10687481)

Mitochondrial reactive oxygen species (mitoROS) assay using H9c2 overexpressing human TFAM (hTFAM) or rat Twinkle-Flag (rTwinkle) and mitochondria from TFAM mice and Twinkle (TW) m... by Masataka Ikeda (428186)

Image_2_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

Image_3_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

Image_6_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

Image_1_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

DataSheet_1_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.xls by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

DataSheet_2_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.xls by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

Image_5_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”

Image_4_Monoallelic Heb/Tcf12 Deletion Reduces the Requirement for NOTCH1 Hyperactivation in T-Cell Acute Lymphoblastic Leukemia.jpeg by Diogo F. T. Veiga (12282320)

“…At the molecular level, HEB protein levels are decreased via proteasomal degradation at the leukemic stage, pointing to a reversible loss of function mechanism. …”