Kinetics of PI(3,4,5)P3 hydrolysis by PTEN at different concentrations of YFP-PHGrp1. by Chun Liu (47227)

“…<p>(A) The membrane composition is 0.2% PI(3,4,5)P3 + 5% DOPS + 0.2% PI(4,5)P2 + 94.4% DOPC + 0.2% TR-DHPE and the bulk concentration of YFP-PHGrp1 is 100 nM (square), 150 nM (circle), 300 nM (triangle), and 600 nM (down-pointing triangle), respectively. …”

Items. by Samuel W. Terman (13584382)

“…<div><p>Epilepsy is a common, serious condition. Fortunately, seizure risk decreases with increasing seizure-free time on antiseizure medications (ASMs). …”

Survey feedback. by Samuel W. Terman (13584382)

“…<div><p>Epilepsy is a common, serious condition. Fortunately, seizure risk decreases with increasing seizure-free time on antiseizure medications (ASMs). …”

<i>osm-5</i> acts in AWB to regulate neuronal responses of AWB to IAA, 2-nonanone, and the mixture. by Wenxing Yang (1491607)

“…<b>(B—D)</b> Exposure to IAA increases GCaMP signal of AWB (ON response) and removal of IAA decreases it (OFF response). The <i>osm-5(p813)</i> mutation disrupts both the ON and OFF responses and expressing <i>osm-5</i> in AWB rescues the defects. …”

<i>Klf5</i> overexpression in intestinal epithelia increases STAT3 phosphorylation <i>in vivo</i>. by Marie-Pier Tetreault (320873)

“…(<b>C</b>) Western blot of colonic epithelial scrapings from control (Cn) and <i>Villin-Klf5</i> (Vl) mice at the indicated time points following DSS treatment revealed increased phospho-STAT3 and phospho-JAK2 in <i>Villin-Klf5</i> mice at day 7, while phospho-STAT3 was decreased in <i>Villin-Klf5</i> mice at day 0 and unchanged at day 3. …”

Data Sheet 5_The power of GM-CSF: immune regulation in the defense against Phialophora verrucosa infection.pdf by Qi Dong (119569)

Climate complexity in the migratory cycle of <i>Ammodramus bairdii</i> by Alexander Peña-Peniche (5687786)

“…A continuous and alarming decrease of its populations has been observed over the last 50 years, and studying its seasonal distribution and associated climatic niches could help improve strategies for its conservation. …”

Explanatory power of risk measures in long term by diversification. by Mihály Ormos (697939)

S2 Table - by Penny A. Rudd (10298691)

Mst1 knockout skewed macrophage polarization toward a proinflammatory and profibrotic phenotype with concomitant down-regulation of CD36. by Jianyang Li (461703)

HCV elicits differential expression of circRNAs. by Tzu-Chun Chen (787255)

YorkU.forest.Oct25-2016.csv by Andrew Laruffa (3116073)

“…</p><p>The methods are as follows:</p><p>-A transect measuring 50m was randomly placed.</p><p>-For every two meters along the transect:</p><p>·      The number of woody plants was counted on either side of the transect that were within 0.5 meters.…”

Blockade of IFN<i>γ</i> production rescues the T_reg cell homeostatic defect and alleviates systemic autoimmunity in <i>Foxp3</i>^<i>Cre</i><i>miR-142</i><sup><... by Wei-Le Wang (133402)

Secondary attack rates among 1407 close contacts based on case-patient^a characteristics, their own characteristics, and exposure setting. by Joseph Walker (4341838)

Data_Sheet_5_Bacteroides abundance drives birth mode dependent infant gut microbiota developmental trajectories.XLSX by Dollwin Matharu (13914927)

“…Background and aims<p>Birth mode and other early life factors affect a newborn's microbial colonization with potential long-term health effects. …”

Mendelian randomization results with COVID-19. by Alish B. Palmos (5857379)

Schematic diagram of STTFP model. by Guozhu Sui (21672798)

PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”