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step decrease » sizes decrease (Expand Search), teer decrease (Expand Search), we decrease (Expand Search)
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a step » _ step (Expand Search)
step decrease » sizes decrease (Expand Search), teer decrease (Expand Search), we decrease (Expand Search)
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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1961
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1962
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1963
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1964
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1965
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1966
DataSheet1.docx
Published 2018“…Interestingly, host contact increased the expression and secretion of V. vulnificus RtxA1 toxin, which was decreased and delayed by the rpoS mutation. …”
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1967
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1968
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1969
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1970
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1971
Tannic acid blocks an early step of HCV entry.
Published 2015“…Huh7.5 cells were inoculated with HCV JFH1-AM120-Rluc at a MOI of 10 for one hour at 4°C in presence of 50 μM of tannic acid, 500 μg/ml of heparin or no inhibitor as a control. …”
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1972
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1973
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1974
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1975
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1976
<i>Otx</i> knockdown causes a decrease in <i>dct</i> expression in the developing RPE.
Published 2012“…<i>dct</i> expression was reduced only in the ventral most region of the optic cup in single morphants (B–D, white arrows) compared to controls (A). <i>dct</i> expression was significantly decreased in the ventral RPE of <i>otx1a</i>/<i>otx2</i> morphants and to an even greater degree in <i>otx1a</i>/<i>otx1b</i>/<i>otx2</i> morphants (E–F, white arrows). …”
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1977
Image3_Biochanin A Regulates Key Steps of Inflammation Resolution in a Model of Antigen-Induced Arthritis via GPR30/PKA-Dependent Mechanism.TIF
Published 2021“…BCA treatment shortened Ri from ∼23 h observed in vehicle-treated mice to ∼5.5 h, associated with an increase in apoptotic events and efferocytosis, both key steps for the resolution of inflammation. These effects of BCA were prevented by H89, an inhibitor of protein kinase A (PKA) and G15, a selective G protein–coupled receptor 30 (GPR30) antagonist. …”
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1978
Image2_Biochanin A Regulates Key Steps of Inflammation Resolution in a Model of Antigen-Induced Arthritis via GPR30/PKA-Dependent Mechanism.TIF
Published 2021“…BCA treatment shortened Ri from ∼23 h observed in vehicle-treated mice to ∼5.5 h, associated with an increase in apoptotic events and efferocytosis, both key steps for the resolution of inflammation. These effects of BCA were prevented by H89, an inhibitor of protein kinase A (PKA) and G15, a selective G protein–coupled receptor 30 (GPR30) antagonist. …”
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1979
Image4_Biochanin A Regulates Key Steps of Inflammation Resolution in a Model of Antigen-Induced Arthritis via GPR30/PKA-Dependent Mechanism.TIF
Published 2021“…BCA treatment shortened Ri from ∼23 h observed in vehicle-treated mice to ∼5.5 h, associated with an increase in apoptotic events and efferocytosis, both key steps for the resolution of inflammation. These effects of BCA were prevented by H89, an inhibitor of protein kinase A (PKA) and G15, a selective G protein–coupled receptor 30 (GPR30) antagonist. …”
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1980
Image1_Biochanin A Regulates Key Steps of Inflammation Resolution in a Model of Antigen-Induced Arthritis via GPR30/PKA-Dependent Mechanism.TIF
Published 2021“…BCA treatment shortened Ri from ∼23 h observed in vehicle-treated mice to ∼5.5 h, associated with an increase in apoptotic events and efferocytosis, both key steps for the resolution of inflammation. These effects of BCA were prevented by H89, an inhibitor of protein kinase A (PKA) and G15, a selective G protein–coupled receptor 30 (GPR30) antagonist. …”