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we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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19681
Table1_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.docx
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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19682
DataSheet4_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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19683
Table2_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.docx
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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19684
DataSheet6_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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19685
DataSheet2_NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis.zip
Published 2023“…Meanwhile, the glycolysis rate of drug-resistant cancer cells has increased. NOP58 knockdown decreased glycolysis and enhanced the sensitivity of 116-5FuR and Lovo-5FuR cells to 5FU.…”
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19686
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19687
Sodium butyrate alleviates potential Alzheimer’s disease in vitro by suppressing Aβ and tau activation and ameliorates Aβ-induced toxicity
Published 2023“…Sodium butyrate (NaB) is an HDAC inhibitor profoundly found to be neuroprotective. We have investigated the neuroprotective effects of NaB in SH-SY5Y cells stimulated with TNF-α/Aβ in SH-SY5Y cells and LPS-induced BV-2 cells. …”
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19688
Unveiling the Cytotoxic Potential of Quercetin-Loaded Magnetic Bacterial Bots against Cervical Cancer
Published 2025“…The cytotoxicity of the fabricated magnetic bacterial bots within the low dose (5 × 10<sup>5</sup> CFU/mL) against the HeLa monolayer and 3D spheroid model was found around 84%, which had remarkably enhanced up to more than 91% in the presence of an external magnet. …”
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19689
Unveiling the Cytotoxic Potential of Quercetin-Loaded Magnetic Bacterial Bots against Cervical Cancer
Published 2025“…The cytotoxicity of the fabricated magnetic bacterial bots within the low dose (5 × 10<sup>5</sup> CFU/mL) against the HeLa monolayer and 3D spheroid model was found around 84%, which had remarkably enhanced up to more than 91% in the presence of an external magnet. …”
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19690
Unveiling the Cytotoxic Potential of Quercetin-Loaded Magnetic Bacterial Bots against Cervical Cancer
Published 2025“…The cytotoxicity of the fabricated magnetic bacterial bots within the low dose (5 × 10<sup>5</sup> CFU/mL) against the HeLa monolayer and 3D spheroid model was found around 84%, which had remarkably enhanced up to more than 91% in the presence of an external magnet. …”
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19691
Table4_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19692
Table2_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19693
Table3_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19694
Image6_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19695
Table1_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.DOCX
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19696
Image1_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19697
Image4_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19698
Image2_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19699
Image3_SEC5 is involved in M2 polarization of macrophages via the STAT6 pathway, and its dysfunction in decidual macrophages is associated with recurrent spontaneous abortion.JPEG
Published 2022“…We subsequently found that SEC5 interacted with STAT6 in THP-1-derived Mϕs. …”
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19700