Table_1_Immunoglobulin G Subclass-Specific Glycosylation Changes in Primary Epithelial Ovarian Cancer.DOCX by Marta Wieczorek (8846066)

“…In order to separate IgG₂ and IgG₃, we performed a two-step affinity purification employing Protein A and Protein G Sepharose. …”

Image_2_Immunoglobulin G Subclass-Specific Glycosylation Changes in Primary Epithelial Ovarian Cancer.JPEG by Marta Wieczorek (8846066)

“…In order to separate IgG₂ and IgG₃, we performed a two-step affinity purification employing Protein A and Protein G Sepharose. …”

Image_1_Immunoglobulin G Subclass-Specific Glycosylation Changes in Primary Epithelial Ovarian Cancer.jpg by Marta Wieczorek (8846066)

“…In order to separate IgG₂ and IgG₃, we performed a two-step affinity purification employing Protein A and Protein G Sepharose. …”

Table_4_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.xlsx by Victoria Sun (11195334)

Image_1_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.pdf by Victoria Sun (11195334)

Image_4_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.pdf by Victoria Sun (11195334)

Image_5_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.pdf by Victoria Sun (11195334)

Table_2_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.xlsx by Victoria Sun (11195334)

Image_3_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.pdf by Victoria Sun (11195334)

Table_3_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.xlsx by Victoria Sun (11195334)

Table_1_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.xlsx by Victoria Sun (11195334)

Image_2_The Metabolic Landscape of Thymic T Cell Development In Vivo and In Vitro.pdf by Victoria Sun (11195334)

Postsynaptic factors of the learning rule. by Eleni Vasilaki (33913)

“…<p>A model neuron receives constant strong input making it fire at about 50Hz. …”

QRT-PCR for 27-hydroxylase and ABCA1 message in NS398-treated THP-1 cells exposed to prostaglandins by Edwin SL Chan (28334)

“…THP-1 human monocytes were exposed to the following conditions represented by the six bars (from left to right): (1) RPMI 1640, (2) NS398 (50 μM), (3) PGE(0.1 μM) + NS398 (50 μM), (4) PGE(0.1 μM) + NS398 (50 μM), (5) PGE(0.1 μM) + PGE(0.1 μM) + NS398 (50 μM), and (6) PGD(14 μM) + NS398 (50 μM) (all 18-hour exposures). …”

QRT-PCR for 27-hydroxylase and ABCA1 message in NS398-treated THP-1 macrophages exposed to prostaglandins or TXA by Edwin SL Chan (28334)

“…THP-1 human macrophages were exposed to the following conditions represented by the five bars (from left to right): (1) RPMI 1640, (2) NS398 (50 μM), (3) PGE(0.1 μM) + NS398 (50 μM), (4) PGD(14 μM) + NS398 (50 μM), and (5) TXA(3 μM) + NS398 (50 μM) (24-hour exposures to NS398 alone followed by addition of indicated PG or TXAfor a further 24 hours). …”

Toxins from the Caribbean sea anemone Bunodeopsis globulifera increase cisplatin-induced cytotoxicity of lung adenocarcinoma cells by Heidi I Monroy-Estrada (5669477)

“…Results Pre-exposure to CV, F1 and F2 fractions increases cisplatin cytotoxicity in human lung adenocarcinoma cells under specific conditions. Exposure to CV at 50 μgmL-1 induced a reduction of approximately 50% in cell viability, while a similar cytotoxic effect was observed when cell culture was exposed to F1 at 25 μgmL -1 or F2 at 50 μgmL-1. …”

The impact of outcome perceptibility on the precision of the estimate of the differential effect of treatment. by Ognjen Arandjelović (295373)

G-CSF knockdown reduces MDSC accumulation. by Jeremy D. Waight (195980)

Supplementary figure 1 by Cass Dedert (12837572)

“…</strong>Primary cortical neurons were incubated in Neurobasal medium containing either 25 mM (control), 50 mM, or 100 mM glucose. After 72 hours of treatment, viability showed a dose-dependent decrease as glucose concentration increased. …”

Anchusa italica extract: phytochemical and neuroprotective evaluation on global cerebral ischemia and reperfusion by Akram Torki (5383154)

“…Induction of ischemia/reperfusion caused a decrease in the antioxidant capacity of the brain and serum as well as an increase in the malondialdehyde of the brain and serum. …”