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point decrease » point increase (Expand Search)
we decrease » _ decrease (Expand Search), mean decrease (Expand Search), teer decrease (Expand Search)
nn decrease » _ decrease (Expand Search), mean decrease (Expand Search), gy decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
5 point » _ point (Expand Search)
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8241
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8242
Data_Sheet_5_Bacteroides abundance drives birth mode dependent infant gut microbiota developmental trajectories.XLSX
Published 2022“…Background and aims<p>Birth mode and other early life factors affect a newborn's microbial colonization with potential long-term health effects. …”
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8243
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8244
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8245
PCAIs inhibit NCI-H23 cell line viability.
Published 2024“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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8246
PCAIs disrupt actin filaments in NCI-H23 cells.
Published 2024“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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8247
PCAIs suppress 3D NCI-H23 cell invasion.
Published 2024“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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8248
PCAIs suppress NCI-H23 cancer cell migration.
Published 2024“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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8249
PCAIs induce apoptosis in NCI-H23 cells.
Published 2024“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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8250
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8251
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8252
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8253
DNMT1 reduces cisplatin sensitivity partially through downregulating FOXO3a in ovarian cancer cells
Published 2025“…Knocking down of DNMT1 could decrease the IC<sub>50</sub> of cisplatin. Treatment with cisplatin may reduce FOXO3a expression in OC cells. …”
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8254
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8255
A randomized controlled trial of a combination of antiviral and nonsteroidal anti-inflammatory treatment in a bovine model of respiratory syncytial virus infection
Published 2020“…This benefit was greater when treatment was initiated at 3 days rather than 5 days post infection with decreased clinical scores and lower respiratory rates at both time points. …”
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8256
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8257
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8258
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8259
Suppression by extracellular QRNA from TNP Ts Sup free of exosomes, or miR-150 alone, associating with B-cell derived Ag-specific exosomes from the assayed CS-effector cell mixture...
Published 2015“…</b></u> Similarly, two day immune B-1 B cell-derived exosomes, supplemented with decreasing doses of miR-150 alone, mediated suppression of TNP-CS-effector cell adoptive transfer (Groups E-G), down to a dose of 750pg per eventual recipient, which is 50 femtomoles per eventual recipient (Group G). …”
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8260