Mst1 knockout skewed macrophage polarization toward a proinflammatory and profibrotic phenotype with concomitant down-regulation of CD36. by Jianyang Li (461703)

Bidimensional High-Low Frequency (HF, LF) plane representation of the interaction among Phase and High-Low Clarity Groups_: In the Low Clarity (LC) group there was a larg... by David Vagni (8060432)

Contribution of nitric oxide and NADPH oxidase to the vascular effects of angiotensin. by Graziela N. Hagihara (623403)

HCV elicits differential expression of circRNAs. by Tzu-Chun Chen (787255)

Blockade of IFN<i>γ</i> production rescues the T_reg cell homeostatic defect and alleviates systemic autoimmunity in <i>Foxp3</i>^<i>Cre</i><i>miR-142</i><sup><... by Wei-Le Wang (133402)

Secondary attack rates among 1407 close contacts based on case-patient^a characteristics, their own characteristics, and exposure setting. by Joseph Walker (4341838)

Effect of obesity on contraction of mesenteric arteries to angiotensin II and norepinephrine. by Graziela N. Hagihara (623403)

Involvement of MAPKs on angiotensin II-induced contraction. by Graziela N. Hagihara (623403)

Mendelian randomization results with COVID-19. by Alish B. Palmos (5857379)

Schematic diagram of STTFP model. by Guozhu Sui (21672798)

Rho-mediated control of <i>kinB</i> transcription is removed during sporulation. by Vladimir Bidnenko (4258573)

“…The highlighted points correspond to the WT profiles shown in panel (A): LBtran (orange), S1 (green), S5 (blue), S6 (pink); and RM (red) and WT (black) during exponential growth in LB. …”

PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

Expression of CRYAB in normal and tumor tissue samples across different cancer types. by Rasool Saghaleyni (10526301)

Contribution of angiotensin II receptors activation to the vascular effects of angiotensin. by Graziela N. Hagihara (623403)

DNMT1 reduces cisplatin sensitivity partially through downregulating FOXO3a in ovarian cancer cells by Chong Guo (5819105)

“…Knocking down of DNMT1 could decrease the IC₅₀ of cisplatin. Treatment with cisplatin may reduce FOXO3a expression in OC cells. …”

Efficiency of explaining and predicting risk premium in different samples. by Mihály Ormos (697939)