Video example of a boundary vector cell. by Lear Cohen (15353793)

Elimination of the MRC rescues the prophase I delay due to constitutive Rad51 activation, while decreasing spore viability and increasing MI nondisjunction. by Andrew Ziesel (9422868)

Illustration of the difference between the IC50 value assessed directly from experimental data and model-based deconvolution of mechanisms explaining downstream inhibition. by Jennifer L. Wilson (6061124)

“…In the proposed modeling framework, it is possible to separate effects on a specific cell type, from propagated effects, such as loss of upstream progenitors. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Self-Repair of Structure and Bioactivity in a Supramolecular Nanostructure by Charlotte H. Chen (5915282)

“…Thermal energy in this cycle enables noncovalent interactions to reconfigure the nanostructures into the thermodynamically preferred long nanofibers, a repair process that is impeded by kinetic traps. …”

Cell cycle accumulation at G2/M in MTDH knockdown Hec50co cells at 24 hours after LBH589 or LBH589 and TRAIL treatment. by Xiangbing Meng (200278)

“…<p>(A) Cell cycle profiles were determined in control or MTDH knockdown Hec50co cells at 24 hours after treatment with 20 nM LBH589, 25 ng/ml TRAIL, or LBH589 and TRAIL in combination. …”

Oxidation of L-[1-¹⁴C]Cys as a percentage of dose in enterally fed piglets receiving graded levels of Cys and 0.25 g kg^-1 d^-1 Met. by Anna K. Shoveller (6208457)

Tryptophan and metabolite alterations in response to a control diet or a diet containing MCOA. by Natalie E. Diether (16705804)

<i>In vitro</i> drug susceptibility of a panel of recent clinical <i>L</i>. <i>infantum</i> isolates against antileishmanial reference drugs in THP-1, bone-marrow derived macrophag... by Sarah Hendrickx (321143)

Preformed biofilm. by Rodrigo Rollin-Pinheiro (239450)

Biofilm formation. by Rodrigo Rollin-Pinheiro (239450)

ET-26 hydrochloride (ET-26 HCl) has similar hemodynamic stability to that of etomidate in normal and uncontrolled hemorrhagic shock (UHS) rats by Bin Wang (30851)

“…Rats in the UHS group underwent experimentally induced UHS with a target arterial pressure of 40 mmHg for 1 hour, followed by administration of an ED₅₀ dose of one of the experimental agents. …”