Showing 13,861 - 13,880 results of 49,463 for search '(( 50 ((we decrease) OR (a decrease)) ) OR ( 5 ((point decrease) OR (mean decrease)) ))', query time: 0.83s Refine Results
  1. 13861
  2. 13862

    Climate complexity in the migratory cycle of <i>Ammodramus bairdii</i> by Alexander Peña-Peniche (5687786)

    Published 2018
    “…A continuous and alarming decrease of its populations has been observed over the last 50 years, and studying its seasonal distribution and associated climatic niches could help improve strategies for its conservation. …”
  3. 13863
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  5. 13865
  6. 13866
  7. 13867

    PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  8. 13868

    PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  9. 13869

    PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  10. 13870

    PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  11. 13871

    PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  12. 13872

    <i>Klf5</i> overexpression in intestinal epithelia increases STAT3 phosphorylation <i>in vivo</i>. by Marie-Pier Tetreault (320873)

    Published 2013
    “…(<b>C</b>) Western blot of colonic epithelial scrapings from control (Cn) and <i>Villin-Klf5</i> (Vl) mice at the indicated time points following DSS treatment revealed increased phospho-STAT3 and phospho-JAK2 in <i>Villin-Klf5</i> mice at day 7, while phospho-STAT3 was decreased in <i>Villin-Klf5</i> mice at day 0 and unchanged at day 3. …”
  13. 13873
  14. 13874

    The capacity to neutralize free viruses prior to completion of CD4 attachment varies across bnAb classes. by Lucia Reh (546554)

    Published 2015
    “…<p><b>A:</b> Schematic of free virus infection of A3.01-CCR5 target cells to measure total and pre-attachment activity of bnAbs. …”
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