Showing 27,401 - 27,420 results of 27,599 for search '(( 50 ((we decrease) OR (mean decrease)) ) OR ( 50 ((c decrease) OR (a decrease)) ))', query time: 1.08s Refine Results
  1. 27401

    DataSheet1_Tormentic Acid Ameliorates Hepatic Fibrosis in vivo by Inhibiting Glycerophospholipids Metabolism and PI3K/Akt/mTOR and NF-κB Pathways: Based on Transcriptomics and Meta... by Xing Lin (534240)

    Published 2022
    “…The rats were intragastrically administered with 50% CCl<sub>4</sub> for 9 weeks to induce hepatic fibrosis, followed by various agents for 6 weeks. …”
  2. 27402
  3. 27403

    Urine Bacterial Community Convergence through Fertilizer Production: Storage, Pasteurization, and Struvite Precipitation by Rebecca H. Lahr (3217476)

    Published 2016
    “…Urine pasteurization or struvite precipitation did not change the microbial community, even when pasteurized urine was stored for an additional 70 days. Pasteurization decreased metabolic activity by 50 ± 10% and additional storage after pasteurization did not lead to recovery of metabolic activity. …”
  4. 27404

    DataSheet2_Tormentic Acid Ameliorates Hepatic Fibrosis in vivo by Inhibiting Glycerophospholipids Metabolism and PI3K/Akt/mTOR and NF-κB Pathways: Based on Transcriptomics and Meta... by Xing Lin (534240)

    Published 2022
    “…The rats were intragastrically administered with 50% CCl<sub>4</sub> for 9 weeks to induce hepatic fibrosis, followed by various agents for 6 weeks. …”
  5. 27405

    Table_1_Genetic polymorphisms in CYP4F2 may be associated with lung cancer risk among females and no-smoking Chinese population.docx by Hongyang Shi (836187)

    Published 2023
    “…</p>Results<p>This study observed that rs12459936 was linked to an increased risk of LC in no-smoking participants (allele: OR = 1.38, p = 0.035; homozygote: OR = 2.00, p = 0.035; additive: OR = 1.40, p = 0.034) and females (allele: OR = 1.64, p = 0.002; homozygote: OR = 2.57, p = 0.006; heterozygous: OR = 2.56, p = 0.001; dominant: OR = 2.56, p < 0.002; additive: OR = 1.67, p = 0.002). Adversely, there was a significantly decreased LC risk for rs3093110 in no-smoking participants (heterozygous: OR = 0.56, p = 0.027; dominant: OR = 0.58, p = 0.035), rs3093193 (allele: OR = 0.66, p = 0.016; homozygote: OR = 0.33, p = 0.011; recessive: OR = 0.38, p = 0.021; additive: OR = 0.64, p = 0.014), rs3093144 (recessive: OR = 0.20, p = 0.045), and rs3093110 (allele: OR = 0.54, p = 0.010; heterozygous: OR = 0.50, p = 0.014; dominant: OR = 0.49, p = 0.010; additive: OR = 0.54, p = 0.011) in females.…”
  6. 27406

    Image_2_Efficacy and safety of anti-vascular endothelial growth factor agents in the treatment of primary pterygium.JPEG by Bowen Zhang (595078)

    Published 2023
    “…Anti-VEGF agents statistically decreased recurrence rate of pterygium following surgery (RR 0.47, 95% CI 0.31–0.74, P < 0.001). …”
  7. 27407

    Image_1_Efficacy and safety of anti-vascular endothelial growth factor agents in the treatment of primary pterygium.JPEG by Bowen Zhang (595078)

    Published 2023
    “…Anti-VEGF agents statistically decreased recurrence rate of pterygium following surgery (RR 0.47, 95% CI 0.31–0.74, P < 0.001). …”
  8. 27408

    Image_3_Efficacy and safety of anti-vascular endothelial growth factor agents in the treatment of primary pterygium.JPEG by Bowen Zhang (595078)

    Published 2023
    “…Anti-VEGF agents statistically decreased recurrence rate of pterygium following surgery (RR 0.47, 95% CI 0.31–0.74, P < 0.001). …”
  9. 27409

    Image_4_Efficacy and safety of anti-vascular endothelial growth factor agents in the treatment of primary pterygium.JPEG by Bowen Zhang (595078)

    Published 2023
    “…Anti-VEGF agents statistically decreased recurrence rate of pterygium following surgery (RR 0.47, 95% CI 0.31–0.74, P < 0.001). …”
  10. 27410

    Data_Sheet_1_Efficacy and safety of anti-vascular endothelial growth factor agents in the treatment of primary pterygium.PDF by Bowen Zhang (595078)

    Published 2023
    “…Anti-VEGF agents statistically decreased recurrence rate of pterygium following surgery (RR 0.47, 95% CI 0.31–0.74, P < 0.001). …”
  11. 27411

    Image 1_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.tif by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  12. 27412

    Table 3_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.xlsx by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  13. 27413

    DataSheet_1_Based on machine learning algorithms for estimating leaf phosphorus concentration of rice using optimized spectral indices and continuous wavelet transform.pdf by Yi Zhang (9093)

    Published 2023
    “…To obtain the LPC and leaf spectra reflectance, the pot experiments with four phosphorus (P) treatments and two rice cultivars were carried out in a greenhouse in 2020-2021. The results indicated that P deficiency increased leaf reflectance in the visible region (350-750 nm) and decreased the reflectance in the near-infrared (NIR, 750-1350 nm) regions compared to the P-sufficient treatment. …”
  14. 27414

    Data Sheet 4_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.... by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  15. 27415

    Data used in the experiment. by Xuechun Wang (8504442)

    Published 2023
    “…<i>cinerea</i> conidia captured per plate decreased for all mulch treatments. More than 50% and approximately 80% of the total number of dispersed conidia were found on plates 10 and 16 cm away from the inoculum source across all treatments, respectively. …”
  16. 27416

    Table 2_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.xlsx by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  17. 27417

    Table 1_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.xlsx by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  18. 27418

    Table_1_In vitro Analysis of the Intradiscal Pressure of the Thoracic Spine.XLSX by Hans-Joachim Wilke (436691)

    Published 2020
    “…During loading, the IDP was measured using a flexible sensor tube, which was inserted into the nucleus pulposus under x-ray control. …”
  19. 27419

    Data Sheet 1_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.... by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”
  20. 27420

    Data Sheet 2_Polygonatum sibiricum polysaccharides enhance pancreatic β-cell function in diabetic zebrafish by mitigating mitochondrial oxidative damage via the AMPK-SIRT1 pathway.... by Fan Lin (625737)

    Published 2025
    “…Background<p>Mitochondrial oxidative damage in pancreatic β-cells is a key contributor to diabetes pathogenesis, particularly under hyperglycemic conditions. …”