Image_3_Intranasal Delivery of Recombinant S100A8 Protein Delays Lung Cancer Growth by Remodeling the Lung Immune Microenvironment.tif by Sze Wing Wong (1617550)

“…In support of this, we found decreased MDSC numbers, and increased numbers of CD4⁺ T cells and natural killer T (NK-T) cells in lungs from cancer-bearing mice treated with S100A8. …”

Image_2_Intranasal Delivery of Recombinant S100A8 Protein Delays Lung Cancer Growth by Remodeling the Lung Immune Microenvironment.tif by Sze Wing Wong (1617550)

“…In support of this, we found decreased MDSC numbers, and increased numbers of CD4⁺ T cells and natural killer T (NK-T) cells in lungs from cancer-bearing mice treated with S100A8. …”

Image_4_Intranasal Delivery of Recombinant S100A8 Protein Delays Lung Cancer Growth by Remodeling the Lung Immune Microenvironment.tif by Sze Wing Wong (1617550)

“…In support of this, we found decreased MDSC numbers, and increased numbers of CD4⁺ T cells and natural killer T (NK-T) cells in lungs from cancer-bearing mice treated with S100A8. …”

Image_1_Intranasal Delivery of Recombinant S100A8 Protein Delays Lung Cancer Growth by Remodeling the Lung Immune Microenvironment.tif by Sze Wing Wong (1617550)

“…In support of this, we found decreased MDSC numbers, and increased numbers of CD4⁺ T cells and natural killer T (NK-T) cells in lungs from cancer-bearing mice treated with S100A8. …”

A map indicating study areas and locations of samples. by Matti Viluksela (321291)

Table_1_100% Fruit Juice and Dental Health: A Systematic Review of the Literature.docx by DeAnn Liska (6948188)

“…RCTs on tooth erosion showed decreased microhardness, increased surface enamel loss, increased erosion depth, greater enamel softening, and/or increased pellicle layer with 100% fruit juice, and those on dental caries showed increased demineralization of enamel slabs with 100% fruit juice.…”

Calcineurin mediates hyperactive Ste11^{<i>ΔN467</i>}-induced and caspofungin-induced unmasking in <i>C</i>. <i>albicans</i>. by Andrew S. Wagner (11331477)

Trends in the age-standardized rate of YLDs per 100,000 caused by smoking-related strokes in Saudi Arabia during 1990–2021, stratified by gender. by Jaber S. Alqahtani (8822396)

Trends in age-standardized rate of DALYs per 100,000 with caused by smoking-related strokes in Saudi Arabia during 1990–2021, stratified by gender. by Jaber S. Alqahtani (8822396)

Trends in the age-standardized rate of YLLs per 100,000 caused by smoking-related strokes in Saudi Arabia during 1990–2021, stratified by gender. by Jaber S. Alqahtani (8822396)

Trends in age-standardized rate of deaths per 100,000 caused by smoking-related strokes in Saudi Arabia during 1990–2021, stratified by gender. by Jaber S. Alqahtani (8822396)

The top 100 TCs with highest excess deaths from all-cause, CVDs, and RDs. by Wenzhong Huang (538470)

Population standardized emergency department visit rate after self-harm (per 100,000 mid-year population) by age and sex. by Ye Ji Lee (11055903)

Conditional ablation of <i>Ybx1</i> leads to a reduction in cortical progenitor cell pool and a decrease in cortical thickness. by Jian Zhang (1682)

Study design and settings. by Hyun Jin Han (18592254)

“…We observed an increase in the number of acute myeloid leukemia cases, with age-specific incidence rates escalating in older patients. In contrast a long-term decrease from 1.94 to 1.77 per 100,000 individuals was found in the age-standardized incidence rates. …”

Locations, climate conditions (shown as the mean for 1990–2012, as obtained from the China meteorological sharing service system (http://cdc.cma.gov.cn/)), and the initial surficia... by Weiwei Zhang (222964)

Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”