Analysis of human aneurysmal and normal aortic tissues. by Dong Ma (584680)

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Mitochondrial morphology assay. by Dong Ma (584680)

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SMC-specific knockout of Klf5 (smcKlf5^−/−) exacerbates the progression of Ang II–induced AAA by facilitating ROS formation. by Dong Ma (584680)

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The correlation of the mitochondrial dynamics–related genes with Klf5 in mouse VSMCs. by Dong Ma (584680)

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Numerical raw data. by Dong Ma (584680)

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The primers for real-time PCR. by Dong Ma (584680)

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ERK signaling and expression of β-arrestin2 in Ang II–induced mouse AAA models as well as in WT or Klf5^−/− VSMCs. by Dong Ma (584680)

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Demographic and clinical characteristics of patients with AAA. by Dong Ma (584680)

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The expression of eIF5a, Fis1, Pink1, Drp1, Mfn1, and Mtfr1 and the analysis of mitochondrial morphology. by Dong Ma (584680)

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The effect of ML264 (Klf5 inhibitor) on aneurysm formation induced by Ang II infusion in the early and late stages. by Dong Ma (584680)

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Cardiac function assessed by echocardiography in Ang II–infused young (3 months) or old (18 months) WT and smcKlf5^−/− mice. by Dong Ma (584680)

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Klf5 deficiency in mouse VSMCs leads to mitochondrial fission. by Dong Ma (584680)

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ATP assay. by Dong Ma (584680)

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Representative TUNEL- and DAPI-stained sections from the abdominal aortas of young and old WT and smcKlf5^−/− mice following 28 days of Ang II infusion. by Dong Ma (584680)

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eIF5a affects the expression of mitochondrial dynamics–related genes. by Dong Ma (584680)

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Young (3 months) or old (18 months) WT and smcKlf5^−/− mice were infused with Ang II for 28 days. by Dong Ma (584680)

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Klf5 down-regulation leads to human VSMC senescence. by Dong Ma (584680)

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Surface SiO₂ Thickness Controls Uniform-to-Localized Transition in Lithiation of Silicon Anodes for Lithium-Ion Batteries by Manuel Schnabel (4888426)

“…For 2–3 nm SiO₂, SEI formation above 0.1 V is suppressed, but a hold at low or negative voltages can initiate charge transfer whereupon SEI formation and uniform lithiation occur. …”

Inverse relationship between poison exon usage and expression of multiple sodium channels during mouse brain development. by Yuliya Voskobiynyk (9954619)

“…<p>(A) <i>Scn1a</i> transcripts including exon 20N are highly expressed in the developing mouse brain and decrease dramatically after birth (aqua bars), with a corresponding developmental increase in <i>Scn1a</i> expression (blue bars). …”

Statistically significant union pathways refer to the miRNAs with a decreased expression on day 1. by Morgana Lüdtke Azevedo (15285409)