Showing 2,741 - 2,760 results of 24,497 for search '(( 59 ((0 decrease) OR (a decrease)) ) OR ( 50 ((we decrease) OR (mean decrease)) ))', query time: 1.04s Refine Results
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    Classic lipid profile at baseline (T0) and after six months (T6). by Zahra Lotfollahi (13531402)

    Published 2022
    “…<p>(A) Total cholesterol at T0 and at T6 with combined therapy. …”
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    Nonlatching positive feedback substantially dampens the Poissonian noise-mean inverse relationship, allowing stochastic extinction despite increasing mean-expression levels. by Brandon S. Razooky (816542)

    Published 2017
    “…<p>(A) In the classical Poisson or super-Poissonian transcriptional burst models [<a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.2000841#pbio.2000841.ref050" target="_blank">50</a>], the expression mean scales with variance (σ<sup>2</sup> ∝ μ) such that the noise magnitude (CV<sup>2</sup> = σ<sup>2</sup> / μ<sup>2</sup>) decreases proportionally to the inverse of the mean. …”
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    Image_1_Samm50 Promotes Hypertrophy by Regulating Pink1-Dependent Mitophagy Signaling in Neonatal Cardiomyocytes.TIF by Ran Xu (316855)

    Published 2021
    “…However, its molecular mechanisms underlying pathogenesis remain poorly understood. Here, we studied the function of Samm50 in mitophagy during Ang II-induced cardiomyocyte hypertrophy via lentiviruses mediated knockdown and overexpression of Samm50 protein. …”
  16. 2756

    Table_1_Samm50 Promotes Hypertrophy by Regulating Pink1-Dependent Mitophagy Signaling in Neonatal Cardiomyocytes.XLSX by Ran Xu (316855)

    Published 2021
    “…However, its molecular mechanisms underlying pathogenesis remain poorly understood. Here, we studied the function of Samm50 in mitophagy during Ang II-induced cardiomyocyte hypertrophy via lentiviruses mediated knockdown and overexpression of Samm50 protein. …”
  17. 2757

    Table_2_Samm50 Promotes Hypertrophy by Regulating Pink1-Dependent Mitophagy Signaling in Neonatal Cardiomyocytes.XLSX by Ran Xu (316855)

    Published 2021
    “…However, its molecular mechanisms underlying pathogenesis remain poorly understood. Here, we studied the function of Samm50 in mitophagy during Ang II-induced cardiomyocyte hypertrophy via lentiviruses mediated knockdown and overexpression of Samm50 protein. …”
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