Splenic DCs of the oxATP recipients have decreased ability to produce IL-23 and IL-6 and were poor stimulators for Th17 autoreactive T cells. by Ronglan Zhao (658986)

Analysis of mitochondrial morphology in Notch mutants and Notch signaling in Opa1 depleted type II NB lineages. by Dnyanesh Dubal (12085942)

Proliferation defects in Opa1 depleted type II NB lineages are suppressed by overexpression of Drp1^SD. by Dnyanesh Dubal (12085942)

“…B,F:Representative confocal images of type II NB lineages (mCD8-GFP, green) (yellow dotted line) show decreased number of EdU (red) positive cells per lineage (B) in <i>opa1</i> RNAi. …”

Drp1^SD overexpression in <i>opa1</i> RNAi and <i>marf</i> RNAi expressing type II NB lineages suppresses the differentiation defect. by Dnyanesh Dubal (12085942)

Analysis of change in mature INPs, GMCs and neurons in the type II NB lineage on depletion of mitochondrial morphology proteins. by Dnyanesh Dubal (12085942)

Drp1^SD overexpression partially alleviates the differentiation defect seen in <i>notch</i> RNAi expressing type II NB lineages. by Dnyanesh Dubal (12085942)

Analysis of proliferation in NBs depleted of Opa1 and Marf. by Dnyanesh Dubal (12085942)

Opa1 and Marf depletion leads to fragmented mitochondria and overexpression of Drp1^SD leads to clustered mitochondria in type II NBs. by Dnyanesh Dubal (12085942)

Drp1^SD overexpression in Opa1 or Marf depleted type II NBs rescues the reduced mitochondrial membrane potential. by Dnyanesh Dubal (12085942)

“…Scale bar- 10μm (A). Graph showing normalized TMRM intensities in +/+ (17 type II NBs, 8 brains), Drp1^SD (9,5), <i>opa1</i> RNAi (16,5), Drp1^SD;<i>opa1</i> RNAi 7,4), <i>marf</i> RNAi (11,5), Drp1^SD;<i>marf</i> RNAi (6,4) (B). …”

Depletion of Opa1 shows increased ROS levels in the type II NB lineage. by Dnyanesh Dubal (12085942)

Downregulation of Opa1 and Marf does affect type II NB numbers but affect differentiated cells in lineage. by Dnyanesh Dubal (12085942)

Depletion of mitochondrial fusion proteins Opa1 and Marf result in mitochondrial fragmentation. by Dnyanesh Dubal (12085942)

Expression of mRFP does not perturb the severity of <i>opa1</i> RNAi and <i>marf</i> RNAi phenotypes. by Dnyanesh Dubal (12085942)

Notch signaling regulates fused mitochondrial morphology in INPs and muscle cells. by Dnyanesh Dubal (12085942)

Anion-Assisted Glycosylation of Galactose: A Computational Study by Kerli Tali (22769704)

Flow chart of the expression-based functional screen, with validation steps. by Mohamad Karaky (8910497)

Subjects:

Sensitivity of the PCR for decreasing concentrations of genomic DNA purified from <i>Veillonella</i> species using species-specific primer sets. by Izumi Mashima (3165120)

Major steps of the polyamine metabolism in <i>E. coli</i>. by Rosaria Campilongo (626526)

TUDCA decreases MPO with improved mitochondrial biogenesis, mitochondria-ER contact, and alveolar formation of HOX neonatal rat lungs. by Kirkwood A. Pritchard Jr. (13449794)

“…<p>(<b>A</b>) The increased MPO levels in HOX neonatal lungs are decreased by TUDCA treatment (0.1±0.1-fold; n = 6, p<0.05). …”

FASN inhibitors result in decreased ΔΨm and increased superoxide production in melan-a cells. by Franco A. Rossato (588513)