Image1_Therapeutic Effects of Kefir Peptides on Hemophilia-Induced Osteoporosis in Mice With Deficient Coagulation Factor VIII.TIF by Chih-Ching Yen (199298)

“…The therapeutic effects of KPs were shown in the femoral trabeculae and the 4^th lumbar vertebrae, which increased the trabecular bone mineral density (BMD), bone volume (Tb.BV/TV), and trabecular number (Tb.N) and decreased the trabecular separation (Tb.Sp), and they were also observed in the femoral cortical bones, in which the mechanical properties were enhanced in a dose-dependent manner. …”

Table_4_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Image_1_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.TIF by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Image4_Dipyridamole activates adenosine A2B receptor and AMPK/cAMP signaling and promotes myogenic differentiation of myoblastic C2C12 cells.tif by Miguel Marco-Bonilla (16970271)

“…All adenosine receptors were expressed during differentiation with dipyridamole, increasing A2B expression. Tenofovir maintained inactive AMPK and decreased cAMP levels, as well as PKAα and pCREB expression, which were recovered with dipyridamole.…”

Table_6_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Image3_Dipyridamole activates adenosine A2B receptor and AMPK/cAMP signaling and promotes myogenic differentiation of myoblastic C2C12 cells.tif by Miguel Marco-Bonilla (16970271)

“…All adenosine receptors were expressed during differentiation with dipyridamole, increasing A2B expression. Tenofovir maintained inactive AMPK and decreased cAMP levels, as well as PKAα and pCREB expression, which were recovered with dipyridamole.…”

Table_3_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Image2_Dipyridamole activates adenosine A2B receptor and AMPK/cAMP signaling and promotes myogenic differentiation of myoblastic C2C12 cells.tif by Miguel Marco-Bonilla (16970271)

“…All adenosine receptors were expressed during differentiation with dipyridamole, increasing A2B expression. Tenofovir maintained inactive AMPK and decreased cAMP levels, as well as PKAα and pCREB expression, which were recovered with dipyridamole.…”

Image1_Dipyridamole activates adenosine A2B receptor and AMPK/cAMP signaling and promotes myogenic differentiation of myoblastic C2C12 cells.tif by Miguel Marco-Bonilla (16970271)

“…All adenosine receptors were expressed during differentiation with dipyridamole, increasing A2B expression. Tenofovir maintained inactive AMPK and decreased cAMP levels, as well as PKAα and pCREB expression, which were recovered with dipyridamole.…”

Table_7_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Image5_Dipyridamole activates adenosine A2B receptor and AMPK/cAMP signaling and promotes myogenic differentiation of myoblastic C2C12 cells.tif by Miguel Marco-Bonilla (16970271)

“…All adenosine receptors were expressed during differentiation with dipyridamole, increasing A2B expression. Tenofovir maintained inactive AMPK and decreased cAMP levels, as well as PKAα and pCREB expression, which were recovered with dipyridamole.…”

Phase 1 study to evaluate the effects of rifampin or itraconazole on the pharmacokinetics of limertinib (ASK120067), a novel mutant-selective inhibitor of the epidermal growth fact... by Bei Cao (141848)

“…<p>Limertinib is a novel mutant-selective and irreversible inhibitor of the epidermal growth factor receptor under development. …”

Table_5_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Table_1_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

Table_2_m6A Modification Mediates Mucosal Immune Microenvironment and Therapeutic Response in Inflammatory Bowel Disease.DOC by Yongyu Chen (8477316)

“…<p>Accumulating evidence links m⁶A modification with immune infiltration. However, the correlation and mechanism by which m⁶A modification promotes intestinal immune infiltration in inflammatory bowel disease (IBD) is unknown. …”

DataSheet_1_Efficacy of Flash Glucose Monitoring in Type 1 and Type 2 Diabetes: A Systematic Review and Meta-Analysis of Randomised Controlled Trials.zip by Bonnie Liang (12116082)

“…</p>Conclusions<p>Flash glucose monitoring did not lead to a significant reduction in HbA_1c compared to self-monitoring of blood glucose, however, it improved glycaemic management through increased time in range and decreased frequency of hypoglycaemic episodes.…”

Linear and back-splicing alterations correlate with mis-regulation of post-transcriptional regulators. by Dilara Ayyildiz (17145143)

“…<p>The schematic (created using <a href="http://Biorender.com" target="_blank">Biorender.com</a>) presents a possible model of action of mutant huntingtin, affecting directly or indirectly (through miRNAs) the expression of post-transcriptional regulators, eventually leading to increased alternative linear splicing and decreased circRNAs biogenesis. …”

Latencies are linear functions of pheromone dose with different parameter values in each neuron. by Jean-Pierre Rospars (44871)

“…<p>(<b>A</b>) Measured latency <i>L</i> (dots) of 3 ORNs fitted to decreasing lines (eq. …”

Metformin attenuates multiple myeloma cell proliferation and encourages apoptosis by suppressing METTL3-mediated m6A methylation of THRAP3, RBM25, and USP4 by Cong-Jie Chen (14598552)

“…The effect on MM cell xenograft tumor growth was observed by injection of metformin or/and overexpression of METTL3 in in vivo experiments. Metformin decreased cell proliferation and encouraged cell apoptosis in a dose-dependent manner. …”

Supplementary Material for: Modelling Childhood Growth Using Fractional Polynomials and Linear Splines by Tilling K. (4145887)

“…Here, we demonstrate multilevel models for childhood growth either as a smooth function (using fractional polynomials) or a set of connected linear phases (using linear splines). …”