Data_Sheet_1_Spatial Distribution, Diversity, and Activity of Microbial Phototrophs in the Baltic Sea.pdf by Peihang Xu (8279931)

“…The Baltic Sea is exposed to multiple climate change related stressors, such as warming, decreasing salinity, and deoxygenation. This study contributes to understanding and interpretation of how microbial community, especially phototrophic groups, might shift in their distribution and activity in a changing ocean like the Baltic Sea.…”

CreA maintains fungal bioenergetics homeostasis. by Sarah R. Beattie (3935237)

“…The ratio of each metabolite in Δ<i>creA</i>/WT is given under metabolite name. Metabolites that are boxed in red and green are significantly increased and decreased, respectively, in Δ<i>creA</i>, and black are not significantly changed (p≤0.05 by Welch’s two-sample t-test). mRNA levels of key metabolic enzymes are given as fold-change in Δ<i>creA</i> compared to wild type. …”

Image_2_HOTTIP Predicts Poor Survival in Gastric Cancer Patients and Contributes to Cisplatin Resistance by Sponging miR-216a-5p.JPEG by Rui Zhao (43391)

“…Mechanistic investigations revealed that HOTTIP may regulate the functions of GC cells by sponging miR-216a-5p. MiR-216a-5p overexpression decreased Bcl-2 expression, enhanced Beclin1 expression, and active autophagy. …”

Table_3_HOTTIP Predicts Poor Survival in Gastric Cancer Patients and Contributes to Cisplatin Resistance by Sponging miR-216a-5p.docx by Rui Zhao (43391)

“…Mechanistic investigations revealed that HOTTIP may regulate the functions of GC cells by sponging miR-216a-5p. MiR-216a-5p overexpression decreased Bcl-2 expression, enhanced Beclin1 expression, and active autophagy. …”

Table_1_HOTTIP Predicts Poor Survival in Gastric Cancer Patients and Contributes to Cisplatin Resistance by Sponging miR-216a-5p.docx by Rui Zhao (43391)

“…Mechanistic investigations revealed that HOTTIP may regulate the functions of GC cells by sponging miR-216a-5p. MiR-216a-5p overexpression decreased Bcl-2 expression, enhanced Beclin1 expression, and active autophagy. …”

Data_Sheet_1_DYT1 Dystonia Patient-Derived Fibroblasts Have Increased Deformability and Susceptibility to Damage by Mechanical Forces.PDF by Navjot Kaur Gill (6877559)

“…<p>DYT1 dystonia is a neurological movement disorder that is caused by a loss-of-function mutation in the DYT1/TOR1A gene, which encodes torsinA, a conserved luminal ATPases-associated with various cellular activities (AAA+) protein. …”

Table_2_HOTTIP Predicts Poor Survival in Gastric Cancer Patients and Contributes to Cisplatin Resistance by Sponging miR-216a-5p.docx by Rui Zhao (43391)

“…Mechanistic investigations revealed that HOTTIP may regulate the functions of GC cells by sponging miR-216a-5p. MiR-216a-5p overexpression decreased Bcl-2 expression, enhanced Beclin1 expression, and active autophagy. …”

Image_1_Homer1a Attenuates Endoplasmic Reticulum Stress-Induced Mitochondrial Stress After Ischemic Reperfusion Injury by Inhibiting the PERK Pathway.TIF by Jialiang Wei (1617223)

“…After OGD treatment, the overexpression of Homer1a preserved mitochondrial function, as evidenced by less cytochrome c release, less reactive oxygen species (ROS) production, less ATP and mitochondrial membrane potential (MMP) loss, less caspase-9 activation, and inhibition of endoplasmic reticulum (ER) stress confirmed by the decreased expression of phosphate-PKR-like ER Kinase (p-PERK)/PERK and phosphate- inositol-requiring enzyme 1 (p-IRE1)/IRE1 and immunofluorescence (IF) staining. …”

Image_1_HOTTIP Predicts Poor Survival in Gastric Cancer Patients and Contributes to Cisplatin Resistance by Sponging miR-216a-5p.JPEG by Rui Zhao (43391)

“…Mechanistic investigations revealed that HOTTIP may regulate the functions of GC cells by sponging miR-216a-5p. MiR-216a-5p overexpression decreased Bcl-2 expression, enhanced Beclin1 expression, and active autophagy. …”

Table_7_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

Table_4_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

Presentation1_Ligand-bound integrin αvβ6 internalisation and trafficking.pdf by Amelia Meecham (13301040)

“…<p>The integrin αvβ6 is expressed at low levels in most normal healthy tissue but is very often upregulated in a disease context including cancer and fibrosis. …”

Table_3_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

Table_2_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

DataSheet1_Novel Compounds Synergize With Venetoclax to Target KMT2A-Rearranged Pediatric Acute Myeloid Leukemia.docx by Claudia Tregnago (5518502)

“…Our screening revealed that both the bromodomain and extra-terminal domain (BET) inhibitor, I-BET151, and kinase inhibitor, sunitinib, decreased the BCL-2 family protein expression and significantly synergized with venetoclax, enhancing KMT2A-r AML cell line death. …”

DataSheet1_A Novel Role of PP2A Methylation in the Regulation of Tight Junction Assembly and Integrity.PDF by Diana Schuhmacher (13036623)

“…Here, using MDCK cells, we show that overexpression of LCMT1, which enhances cellular PP2A methylation, inhibits TJ formation, induces TJ ruffling, and decreases TJ barrier function. …”

Table_1_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

Table_6_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

Table_5_The RNA N6-Methyladenosine Methyltransferase METTL3 Promotes the Progression of Kidney Cancer via N6-Methyladenosine-Dependent Translational Enhancement of ABCD1.XLSX by Yue Shi (128342)

“…<p>The role of N6-methyladenosine (m⁶A)-modifying proteins in cancer progression depends on the cell type and mRNA affected. …”

DataSheet2_Novel Compounds Synergize With Venetoclax to Target KMT2A-Rearranged Pediatric Acute Myeloid Leukemia.xlsx by Claudia Tregnago (5518502)

“…Our screening revealed that both the bromodomain and extra-terminal domain (BET) inhibitor, I-BET151, and kinase inhibitor, sunitinib, decreased the BCL-2 family protein expression and significantly synergized with venetoclax, enhancing KMT2A-r AML cell line death. …”