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21321
Data_Sheet_5_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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21322
Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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21323
Image_5_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21324
Image_4_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21325
Image_6_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21326
Table_1_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.xlsx
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21327
Table_3_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.docx
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21328
Image_7_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21329
Image_1_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21330
Table_2_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.docx
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21331
Image_2_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21332
Image_3_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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21333
ARHGEF11 localization and actin (F-actin) cytoskeleton immunofluorescence.
Published 2015“…NRK-shRNA3 cells demonstrate a significant reduction in stress fibers, similar to pharmacological imbibition of ROCK by fasudil, as cell length/width ratio decreases to baseline with genetic knockdown of <i>Arhgef11</i>. …”
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21334
Image3_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.JPEG
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”
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21335
Table1_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.XLSX
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”
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21336
Image1_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.JPEG
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”
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21337
Inhibition of VEGF-A expression by FIH-1.
Published 2014“…Total RNA was obtained and then RQ-PCR was performed using primers specific for VEGF-A and 36B4 (internal control). VEGF-A mRNA levels were decreased with FIH-1 overexpresssion under both normoxic and hypoxic conditions (<a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0086102#pone-0086102-g004" target="_blank">Figure 4A</a>). …”
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21338
Image5_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.JPEG
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”
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21339
Image2_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.JPEG
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”
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21340
Image6_Poly (A) binding protein 2 is critical for stem cell differentiation during regeneration in the planarian Schmidtea mediterranea.JPEG
Published 2024“…A deficit in PABP2 blocks the transition of neoblasts toward immediate early progenitors, leading to an enhanced pool of non-committed neoblasts and a decreased progenitor population. …”