Chromosomal localization and gene evolutionary relationship of <i>PtWRKYs.</i> by Yuzhao Ma (12840632)

Influence of baseline preferences on the implicit measures (spatial indices of mouse kinematics). by Cinzia Calluso (5662420)

Image_2_Baseline Testosterone Predicts Body Composition and Metabolic Response to Testosterone Therapy.jpeg by FNU Deepika (11881310)

“…Men with T ≥ 264 ng/dl showed significant decreases in HbA1c at 12 months (−3.1 ± 9.2 vs. 3.2 ± 13.9%; p = 0.005), fasting glucose at 18 months (−4.2 ± 31.9 vs. 13.0 ± 57.3%; p = 0.040), LDL at 6 months (−6.4 ± 27.5 vs. 12.8 ± 44.1%; p = 0.034), and leptin at 18 months (−40.2 ± 35.1 vs. −27.6 ± 31.0%; p = 0.034) compared to those with T < 264 ng/dl. …”

Image_1_Baseline Testosterone Predicts Body Composition and Metabolic Response to Testosterone Therapy.jpeg by FNU Deepika (11881310)

“…Men with T ≥ 264 ng/dl showed significant decreases in HbA1c at 12 months (−3.1 ± 9.2 vs. 3.2 ± 13.9%; p = 0.005), fasting glucose at 18 months (−4.2 ± 31.9 vs. 13.0 ± 57.3%; p = 0.040), LDL at 6 months (−6.4 ± 27.5 vs. 12.8 ± 44.1%; p = 0.034), and leptin at 18 months (−40.2 ± 35.1 vs. −27.6 ± 31.0%; p = 0.034) compared to those with T < 264 ng/dl. …”

Table_1_Baseline Testosterone Predicts Body Composition and Metabolic Response to Testosterone Therapy.docx by FNU Deepika (11881310)

“…Men with T ≥ 264 ng/dl showed significant decreases in HbA1c at 12 months (−3.1 ± 9.2 vs. 3.2 ± 13.9%; p = 0.005), fasting glucose at 18 months (−4.2 ± 31.9 vs. 13.0 ± 57.3%; p = 0.040), LDL at 6 months (−6.4 ± 27.5 vs. 12.8 ± 44.1%; p = 0.034), and leptin at 18 months (−40.2 ± 35.1 vs. −27.6 ± 31.0%; p = 0.034) compared to those with T < 264 ng/dl. …”

Image 4_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

Image 6_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

Image 5_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

Image 1_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

Image 3_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

Image 2_Chinese massage therapy (Tuina) inhibits motor neuron apoptosis in rats with sciatic nerve injury by regulating the cPLA2 and RhoA/ROCK2 signaling pathways.tif by Jiawei Sun (772107)

“…Objective<p>To investigate whether Tuina therapy alleviated inflammation and motor neuron apoptosis in sciatic nerve injury (SNI) rats by regulating cytosolic phospholipase A2 (cPLA2) and Ras homolog family member A/Rho-associated coiled-coil comprising protein kinase 2 (RhoA/ROCK2) signaling cascades.…”

PtWRKY gene structure and Motif. by Yuzhao Ma (12840632)

<i>PsnWRKY95</i> transgenic plants selection and functional analysis of <i>PsnWRKY95</i> in cadmium resistance. by Yuzhao Ma (12840632)

Analysis of <i>PtWRKYs</i> expression in response to Cd. by Yuzhao Ma (12840632)

C2GnT1 gene knockdown decreases C2-O-sLe^X glycoproteins and C2GnT1 activity. by Catherine A. St. Hill (357388)

“…<p>(A) C2GnT1 suppression by shRNA resulted in decreased mRNA levels of C2GnT1 but not FucT-III in LS174T cells, (scrambled shRNA vector, lane 1, and C2GnT1 shRNA vector, lane 2). …”

Data_Sheet_1_Marathon Running Increases Synthesis and Decreases Catabolism of Joint Cartilage Type II Collagen Accompanied by High-Energy Demands and an Inflamatory Reaction.PDF by José A. Hernández-Hermoso (11545663)

“…Accompanied by an increase in type II collagen cartilage fibrils synthesis (sPIINP increase) and a decrease in its catabolism (sC2C decrease), without changes in non-collagenous cartilage metabolism (sCOMP, sC846, and sYKL-40). …”

Secondary Hypervalent I(III)···O Interactions:  Synthesis and Structure of Hypervalent Complexes of Diphenyl-λ³-iodanes with 18-Crown-6 by Masahito Ochiai (2156452)

“…Substitution of an electron-withdrawing group onto the <i>para</i> position of Ph₂IBF₄ tends to increase in the complex stability. A linear Hammett relationship (ρ = 0.59) between log <i>K</i>_a and σ_p⁺ values of substituents indicates that the diaryl-λ³-iodanes with electropositive iodine(III) interact more efficiently with 18C6. …”

Secondary Hypervalent I(III)···O Interactions:  Synthesis and Structure of Hypervalent Complexes of Diphenyl-λ³-iodanes with 18-Crown-6 by Masahito Ochiai (2156452)

“…Substitution of an electron-withdrawing group onto the <i>para</i> position of Ph₂IBF₄ tends to increase in the complex stability. A linear Hammett relationship (ρ = 0.59) between log <i>K</i>_a and σ_p⁺ values of substituents indicates that the diaryl-λ³-iodanes with electropositive iodine(III) interact more efficiently with 18C6. …”

Secondary Hypervalent I(III)···O Interactions:  Synthesis and Structure of Hypervalent Complexes of Diphenyl-λ³-iodanes with 18-Crown-6 by Masahito Ochiai (2156452)

“…Substitution of an electron-withdrawing group onto the <i>para</i> position of Ph₂IBF₄ tends to increase in the complex stability. A linear Hammett relationship (ρ = 0.59) between log <i>K</i>_a and σ_p⁺ values of substituents indicates that the diaryl-λ³-iodanes with electropositive iodine(III) interact more efficiently with 18C6. …”

Secondary Hypervalent I(III)···O Interactions:  Synthesis and Structure of Hypervalent Complexes of Diphenyl-λ³-iodanes with 18-Crown-6 by Masahito Ochiai (2156452)

“…Substitution of an electron-withdrawing group onto the <i>para</i> position of Ph₂IBF₄ tends to increase in the complex stability. A linear Hammett relationship (ρ = 0.59) between log <i>K</i>_a and σ_p⁺ values of substituents indicates that the diaryl-λ³-iodanes with electropositive iodine(III) interact more efficiently with 18C6. …”