Table_2_Defining the Extracellular Matrix of Rhabdomyosarcoma.xlsx by Xiaolei Lian (10183011)

“…We have uncovered that RMS produces increased expression of type XVIII collagen alpha 1 (COL18A1), which is clinically associated with decreased long-term survival. …”

Image_3_MicroRNA-20a-5p Ameliorates Non-alcoholic Fatty Liver Disease via Inhibiting the Expression of CD36.TIF by Xin Wang (91924)

“…Real-time PCR, dual-luciferase assay, and western blotting were employed to detect the relationship between miR-20a-5p and CD36. The expression level of miR-20a-5p was decreased in NAFLD patients, HFD mice, and free fatty acid (FFA)-treated HepG2 cells or primary mouse hepatocytes, accompanied by increased lipid production in hepatocytes. …”

Image_3_TOP2A correlates with poor prognosis and affects radioresistance of medulloblastoma.tif by Yufeng Zhang (195768)

“…Cell survival curve analysis revealed that the survival fraction of MB cells was significantly reduced upon TOP2A downregulation and that si-TOP2A-transfected cells had decreased D₀, Dq, and SF₂ values, indicating that TOP2A knockdown suppresses the resistance to radiotherapy in MB cells. …”

Image1_Lidocaine Promoted Ferroptosis by Targeting miR-382-5p /SLC7A11 Axis in Ovarian and Breast Cancer.JPEG by Dan Sun (140898)

“…Furthermore, the treatment of lidocaine repressed tumor growth of ovarian cancer cells in vivo, in which the miR-382-5p expression was increased while SLC7A11 expression was decreased. Consequently, we concluded that the lidocaine promoted ferroptosis by miR-382-5p/SLC7A11 axis in ovarian and breast cancer cells. …”

Cell Cycle- and Cancer-Associated Gene Networks Activated by Dsg2: Evidence of Cystatin A Deregulation and a Potential Role in Cell-Cell Adhesion by Abhilasha Gupta (532794)

“…Following microarray and gene network analysis, we demonstrate that Dsg2 caused a profound change in the transcriptome of keratinocytes <i>in vivo</i> and altered a number of genes important in epithelial dysplasia including: calcium-binding proteins (S100A8 and S100A9), members of the cyclin protein family, and the cysteine protease inhibitor cystatin A (CSTA). …”

Table_1_TOP2A correlates with poor prognosis and affects radioresistance of medulloblastoma.docx by Yufeng Zhang (195768)

“…Cell survival curve analysis revealed that the survival fraction of MB cells was significantly reduced upon TOP2A downregulation and that si-TOP2A-transfected cells had decreased D₀, Dq, and SF₂ values, indicating that TOP2A knockdown suppresses the resistance to radiotherapy in MB cells. …”

Data_Sheet_2_Topical Diclofenac Reprograms Metabolism and Immune Cell Infiltration in Actinic Keratosis.PDF by Katrin Singer (431791)

“…</p><p>Methods: This study was designed as a prospective, randomized, controlled, monocentric investigation (ClinicalTrials.gov Identifier: NCT01935531). …”

Image_2_TOP2A correlates with poor prognosis and affects radioresistance of medulloblastoma.tif by Yufeng Zhang (195768)

“…Cell survival curve analysis revealed that the survival fraction of MB cells was significantly reduced upon TOP2A downregulation and that si-TOP2A-transfected cells had decreased D₀, Dq, and SF₂ values, indicating that TOP2A knockdown suppresses the resistance to radiotherapy in MB cells. …”

Data_Sheet_2_Hepatoprotective Effect of San-Cao Granule on Con A-Induced Liver Injury in Mice and Mechanisms of Action Exploration.PDF by YuXue Yang (5369768)

“…Then, the pharmacological study was performed to evaluate the therapeutic effect and the underlying mechanisms of SCG on Con A-induced liver injury in mice.</p><p>Results: This research demonstrated the pharmacological effect of SCG on Con A-induced liver injury, which was through improving the liver function, relieving the pathological changes of liver tissue, decreasing the level of pro-inflammatory cytokines, and thus balancing the pro- and anti-inflammatory cytokines. …”

Data_Sheet_1_Topical Diclofenac Reprograms Metabolism and Immune Cell Infiltration in Actinic Keratosis.PDF by Katrin Singer (431791)

“…</p><p>Methods: This study was designed as a prospective, randomized, controlled, monocentric investigation (ClinicalTrials.gov Identifier: NCT01935531). …”

PTX ameliorates EMT and upregulates miR-140 in A549 cells. by Congjie Wang (446610)

“…**<i>P</i><0.05 versus TGF-β1-treated cells. In <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0070725#pone-0070725-g001" target="_blank">Fig. 1 B, C</a>, PTX treatment resulted in a reversal of TGF-β1 action by upregulating E-cadherin and decreasing vimentin levels. …”

Data_Sheet_1_Hepatoprotective Effect of San-Cao Granule on Con A-Induced Liver Injury in Mice and Mechanisms of Action Exploration.DOCX by YuXue Yang (5369768)

“…Then, the pharmacological study was performed to evaluate the therapeutic effect and the underlying mechanisms of SCG on Con A-induced liver injury in mice.</p><p>Results: This research demonstrated the pharmacological effect of SCG on Con A-induced liver injury, which was through improving the liver function, relieving the pathological changes of liver tissue, decreasing the level of pro-inflammatory cytokines, and thus balancing the pro- and anti-inflammatory cytokines. …”

Image_1_TOP2A correlates with poor prognosis and affects radioresistance of medulloblastoma.tif by Yufeng Zhang (195768)

“…Cell survival curve analysis revealed that the survival fraction of MB cells was significantly reduced upon TOP2A downregulation and that si-TOP2A-transfected cells had decreased D₀, Dq, and SF₂ values, indicating that TOP2A knockdown suppresses the resistance to radiotherapy in MB cells. …”

Image_1_CircCSPP1 Functions as a ceRNA to Promote Colorectal Carcinoma Cell EMT and Liver Metastasis by Upregulating COL1A1.TIF by Qingyuan Wang (214905)

“…Mechanistically, similar miRNA response elements are shared between circCSPP1 and COL1A1. We demonstrated that circCSPP1 upregulates the mRNA levels of COL1A1, which plays a pivotal role in the process of epithelial–mesenchymal transition (EMT), by competitively binding to miR-193a-5p and preventing miR-193a-5p from decreasing the expression of COL1A1. …”

Table_1_CircCSPP1 Functions as a ceRNA to Promote Colorectal Carcinoma Cell EMT and Liver Metastasis by Upregulating COL1A1.DOCX by Qingyuan Wang (214905)

“…Mechanistically, similar miRNA response elements are shared between circCSPP1 and COL1A1. We demonstrated that circCSPP1 upregulates the mRNA levels of COL1A1, which plays a pivotal role in the process of epithelial–mesenchymal transition (EMT), by competitively binding to miR-193a-5p and preventing miR-193a-5p from decreasing the expression of COL1A1. …”

Mean predicted probabilities of complications in the next 48 h (lines) and number of patients with measurements (bars) in uncomplicated course (blue) and adverse outcome group (pin... by Guiyou Yang (10259597)

Mean predicted probabilities of complications in the next 48 h (lines) and number of patients with measurements (bars) in uncomplicated course (blue) and adverse outcome group (pin... by Guiyou Yang (10259597)

Image1_Complex I deficiency in m.3243A>G fibroblasts is alleviated by reducing NADH accumulation.tif by Tongling Liufu (16830612)

“…Previous studies have indicated that NADH: ubiquinone oxidoreductase (complex I) deficiency accompanied by a decreased nicotinamide adenine dinucleotide (NAD⁺)/reduced NAD⁺ (NADH) ratio may play a pivotal role in the pathogenesis of m.3243A>G mutation.…”

presentation1_Resveratrol Pretreatment Improved Heart Recovery Ability of Hyperglycemic Bone Marrow Stem Cells Transplantation in Diabetic Myocardial Infarction by Down-Regulating... by Fengyun Zhang (1515310)

“…</p><p>RESULTS: This study showed that the viability of BMSCs cultured under hyperglycemic conditions was decreased, the cells became senescent. Besides, an obviously increased in the expression of miR-34a was detected. …”

Image1_Long Noncoding RNA HCG18 Promotes Malignant Phenotypes of Breast Cancer Cells via the HCG18/miR-103a-3p/UBE2O/mTORC1/HIF-1α–Positive Feedback Loop.tif by Xu Liu (276996)

“…Furthermore, as a downstream target of the HCG18/miR-103a-3p/UBE2O/mTORC1 axis, hypoxia-inducible factor 1α transcriptionally promoted HCG18 expression and then formed a positive feedback loop in BC. …”