Showing 32,521 - 32,540 results of 226,405 for search '(( a ((a decrease) OR (linear decrease)) ) OR ( a ((greater decrease) OR (largest decrease)) ))', query time: 1.74s Refine Results
  1. 32521

    Video_4_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.mp4 by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  2. 32522

    Video_3_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.mp4 by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  3. 32523

    Image_1_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.tif by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  4. 32524

    Video_1_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.mp4 by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  5. 32525

    Image_2_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.tif by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  6. 32526

    ODE modeling of WNT5A-EGF/AR signaling and experimental validation. by Zhiwei Ji (493628)

    Published 2019
    “…<p>(<b>A)</b> The effect of WNT5A on 22RV1 cell viability. …”
  7. 32527

    Video_2_Defective Neutrophil Transendothelial Migration and Lateral Motility in ARPC1B Deficiency Under Flow Conditions.mp4 by Lanette Kempers (10899027)

    Published 2021
    “…Here we show that under physiological flow conditions human ARPC1B-deficient neutrophils were able to transmigrate through TNF-α-pre-activated endothelial cells with a decreased efficiency and, once transmigrated, showed definite impairment in subendothelial crawling. …”
  8. 32528

    Immune pathways in <i>Ae</i>. <i>aegypti</i> mosquitoes infected by DENV. by Ju-Mei Wang (6589220)

    Published 2019
    “…<p><b>A</b>, Heatmap of transcriptional variation in expression of immune-related genes (ROS pathways and AMP genes) in DENV-infected mosquitoes. …”
  9. 32529

    Data_Sheet_1_The HDAC6 Inhibitor Trichostatin A Acetylates Microtubules and Protects Axons From Excitotoxin-Induced Degeneration in a Compartmented Culture Model.PDF by Kelsey Hanson (5147819)

    Published 2018
    “…While there were no changes to tyrosinated tubulin or MAP1B, acetylated tubulin was significantly (p < 0.05) decreased by 40% at 6 h post-treatment. To determine whether increasing microtubule acetylation prior to kainic acid exposure could prevent axon fragmentation, we investigated the effect of reducing microtubule deacetylation with the HDAC6 inhibitor, trichostatin A. …”
  10. 32530

    Rb and p53 Liver Functions Are Essential for Xenobiotic Metabolism and Tumor Suppression by Sathidpak Nantasanti (3181224)

    Published 2016
    “…We show that loss of Rb and p53 resulted in reduced Cyp3a expression decreased accumulation of protoporphyrin and consequently less ductular reaction in livers of mice fed with DDC for 3 weeks. …”
  11. 32531

    Cells simultaneously expressing insulin, somatostatin and CCKBR are more frequenly found in islets of donors with higher (>42 mmol/mol) HbA<sub>1c</sub>. by Ayelet Lenz (203435)

    Published 2019
    “…Ratio between insulin positive area and somatostatin positive area was decreased in islets of donors with higher HbA<sub>1c</sub>. …”
  12. 32532

    Image_9_Heat-Killed Lacticaseibacillus paracasei GMNL-653 Exerts Antiosteoporotic Effects by Restoring the Gut Microbiota Dysbiosis in Ovariectomized Mice.TIF by Jhih-Hua Jhong (4777659)

    Published 2022
    “…<p>Osteoporosis is a metabolic inflammatory disease, an imbalance occurs between bone resorption and formation, leading to bone loss. …”
  13. 32533

    Table_1_Inhibition of Fatty Acid–Binding Protein 4 Attenuated Kidney Fibrosis by Mediating Macrophage-to-Myofibroblast Transition.XLS by Yanhuan Feng (5005550)

    Published 2020
    “…FABP4 ablation also attenuated the UUO-induced number of MMT cells and serum amyloid A1 (Saa1) expression. The siRNA-mediated Saa1 knockdown decreased the number of MMT cells in vitro. …”
  14. 32534

    Table1_Neutrophil extracellular traps as immunofibrotic mediators in RA-ILD; pilot evaluation of the nintedanib therapy.docx by Aliki I. Venetsanopoulou (12197432)

    Published 2024
    “…Objective<p>Rheumatoid arthritis-associated interstitial lung disease (RA-ILD) is a significant pulmonary complication of RA. This study tried to elucidate the mechanisms enhancing inflammation and causing lung injury in RA-ILD, focusing on the role of neutrophil extracellular traps (NETs). …”
  15. 32535

    Data_Sheet_1_Heat-Killed Lacticaseibacillus paracasei GMNL-653 Exerts Antiosteoporotic Effects by Restoring the Gut Microbiota Dysbiosis in Ovariectomized Mice.XLSX by Jhih-Hua Jhong (4777659)

    Published 2022
    “…<p>Osteoporosis is a metabolic inflammatory disease, an imbalance occurs between bone resorption and formation, leading to bone loss. …”
  16. 32536

    Image1_Neutrophil extracellular traps as immunofibrotic mediators in RA-ILD; pilot evaluation of the nintedanib therapy.tif by Aliki I. Venetsanopoulou (12197432)

    Published 2024
    “…Objective<p>Rheumatoid arthritis-associated interstitial lung disease (RA-ILD) is a significant pulmonary complication of RA. This study tried to elucidate the mechanisms enhancing inflammation and causing lung injury in RA-ILD, focusing on the role of neutrophil extracellular traps (NETs). …”
  17. 32537

    Closing the Loop: Modelling of Heart Failure Progression from Health to End-Stage Using a Meta-Analysis of Left Ventricular Pressure-Volume Loops by David R. Warriner (670445)

    Published 2014
    “…The data demonstrated that, as HF progressed, stroke volume (SV), ejection fraction (EF%) decreased while LV volumes increased. A 2-element lumped parameter model was employed to model the mean loops and the error was calculated between the loops, demonstrating close fit between the loops. …”
  18. 32538

    Data_Sheet_2_Inhibition of Fatty Acid–Binding Protein 4 Attenuated Kidney Fibrosis by Mediating Macrophage-to-Myofibroblast Transition.PDF by Yanhuan Feng (5005550)

    Published 2020
    “…FABP4 ablation also attenuated the UUO-induced number of MMT cells and serum amyloid A1 (Saa1) expression. The siRNA-mediated Saa1 knockdown decreased the number of MMT cells in vitro. …”
  19. 32539

    Data_Sheet_2_Heat-Killed Lacticaseibacillus paracasei GMNL-653 Exerts Antiosteoporotic Effects by Restoring the Gut Microbiota Dysbiosis in Ovariectomized Mice.PDF by Jhih-Hua Jhong (4777659)

    Published 2022
    “…<p>Osteoporosis is a metabolic inflammatory disease, an imbalance occurs between bone resorption and formation, leading to bone loss. …”
  20. 32540

    Data_Sheet_1_Inhibition of Fatty Acid–Binding Protein 4 Attenuated Kidney Fibrosis by Mediating Macrophage-to-Myofibroblast Transition.PDF by Yanhuan Feng (5005550)

    Published 2020
    “…FABP4 ablation also attenuated the UUO-induced number of MMT cells and serum amyloid A1 (Saa1) expression. The siRNA-mediated Saa1 knockdown decreased the number of MMT cells in vitro. …”