BMISES_Data_Part2. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Logistic regression for LSES population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Logistic regression for HSES population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Logistic regression for overall population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

BMISES_Data_Part1. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Baseline characteristics of HSES/LSES population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Baseline characteristics of overall population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Diagram of study population. by Krystal Hunter (6820052)

“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”

Image12_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

Image_4_IRE1-bZIP60 Pathway Is Required for Nicotiana attenuata Resistance to Fungal Pathogen Alternaria alternata.TIF by Zhen Xu (92379)

“…JA signaling pathway is required for N. attenuata resistance to A. alternata. Interestingly, the fungus-elicited transcriptional levels of BiP, PDI, CNX 1-like, CRT, IRE1, and bZIP60(s) were all significantly decreased in JA-deficient or JA-insensitive plants. …”

Image8_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

Image_1_IRE1-bZIP60 Pathway Is Required for Nicotiana attenuata Resistance to Fungal Pathogen Alternaria alternata.TIF by Zhen Xu (92379)

“…JA signaling pathway is required for N. attenuata resistance to A. alternata. Interestingly, the fungus-elicited transcriptional levels of BiP, PDI, CNX 1-like, CRT, IRE1, and bZIP60(s) were all significantly decreased in JA-deficient or JA-insensitive plants. …”

Image11_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

Data_Sheet_1_UGT1A1 Variants c.864+5G>T and c.996+2_996+5del of a Crigler-Najjar Patient Induce Aberrant Splicing in Minigene Assays.PDF by Linda Gailite (8539332)

“…Thus, the three anomalous transcripts are likely to inactivate UGT1A1. Moreover, this patient is also homozygous for the promoter variant A(TA)7TAA that decreases UGT1A1 expression by 70%, so the full-length transcript produced by c.864+5G>T would be even more reduced (<9%), thus supporting the diagnosis of CNS-type II. …”

Table_1_IRE1-bZIP60 Pathway Is Required for Nicotiana attenuata Resistance to Fungal Pathogen Alternaria alternata.XLSX by Zhen Xu (92379)

“…JA signaling pathway is required for N. attenuata resistance to A. alternata. Interestingly, the fungus-elicited transcriptional levels of BiP, PDI, CNX 1-like, CRT, IRE1, and bZIP60(s) were all significantly decreased in JA-deficient or JA-insensitive plants. …”

Image14_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

Image4_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

DataSheet1_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.ZIP by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”

Data_Sheet_2_Mitochondrial C3a Receptor Activation in Oxidatively Stressed Epithelial Cells Reduces Mitochondrial Respiration and Metabolism.PDF by Masaaki Ishii (10259984)

“…Here we tested oxidative stress and intracellular complement in mitochondrial dysfunction in RPE cells using high resolution live-cell imaging, and metabolism analysis in isolated mitochondria using Seahorse technology. While C3aR levels were unaffected by oxidative stress, its cell membrane levels decreased and mitochondrial (mt) localization increased. …”

Image15_Effects of miR-103a-3p Targeted Regulation of TRIM66 Axis on Docetaxel Resistance and Glycolysis in Prostate Cancer Cells.TIF by Qiang Yi (5818526)

“…The DTX-resistant group showed more obvious low expression of miR-103a-3p and high expression of TRIM66. After the DTX-resistant group cells were transfected with miR-103a-3p and TRIM66 plasmids, the expression of miR-103a-3p increased significantly and that of TRIM66 decreased significantly. …”