Table_4_Associations of vitamin D-related single nucleotide polymorphisms with post-stroke depression among ischemic stroke population.XLSX by Dongren Sun (13863590)

“…</p>Results<p>In the dominant, recessive, and over-dominant models, no significant association was observed between the selected SNPs in the CYP24A1 and CYP2R1 genes and PSD. However, univariate and multivariate logistic regression analysis revealed that the CYP27B1 rs10877012 G/G genotype was associated with a decreased risk of PSD (OR: 0.41, 95% CI: 0.18–0.92, p = 0.030 and OR: 0.42, 95% CI: 0.18–0.98, p = 0.040, respectively). …”

Table_5_Associations of vitamin D-related single nucleotide polymorphisms with post-stroke depression among ischemic stroke population.XLSX by Dongren Sun (13863590)

“…</p>Results<p>In the dominant, recessive, and over-dominant models, no significant association was observed between the selected SNPs in the CYP24A1 and CYP2R1 genes and PSD. However, univariate and multivariate logistic regression analysis revealed that the CYP27B1 rs10877012 G/G genotype was associated with a decreased risk of PSD (OR: 0.41, 95% CI: 0.18–0.92, p = 0.030 and OR: 0.42, 95% CI: 0.18–0.98, p = 0.040, respectively). …”

Table_3_Associations of vitamin D-related single nucleotide polymorphisms with post-stroke depression among ischemic stroke population.XLSX by Dongren Sun (13863590)

“…</p>Results<p>In the dominant, recessive, and over-dominant models, no significant association was observed between the selected SNPs in the CYP24A1 and CYP2R1 genes and PSD. However, univariate and multivariate logistic regression analysis revealed that the CYP27B1 rs10877012 G/G genotype was associated with a decreased risk of PSD (OR: 0.41, 95% CI: 0.18–0.92, p = 0.030 and OR: 0.42, 95% CI: 0.18–0.98, p = 0.040, respectively). …”

Data_Sheet_2_Associations of vitamin D-related single nucleotide polymorphisms with post-stroke depression among ischemic stroke population.PDF by Dongren Sun (13863590)

“…</p>Results<p>In the dominant, recessive, and over-dominant models, no significant association was observed between the selected SNPs in the CYP24A1 and CYP2R1 genes and PSD. However, univariate and multivariate logistic regression analysis revealed that the CYP27B1 rs10877012 G/G genotype was associated with a decreased risk of PSD (OR: 0.41, 95% CI: 0.18–0.92, p = 0.030 and OR: 0.42, 95% CI: 0.18–0.98, p = 0.040, respectively). …”

Data_Sheet_1_Associations of vitamin D-related single nucleotide polymorphisms with post-stroke depression among ischemic stroke population.PDF by Dongren Sun (13863590)

“…</p>Results<p>In the dominant, recessive, and over-dominant models, no significant association was observed between the selected SNPs in the CYP24A1 and CYP2R1 genes and PSD. However, univariate and multivariate logistic regression analysis revealed that the CYP27B1 rs10877012 G/G genotype was associated with a decreased risk of PSD (OR: 0.41, 95% CI: 0.18–0.92, p = 0.030 and OR: 0.42, 95% CI: 0.18–0.98, p = 0.040, respectively). …”

The E3 Ubiquitin Ligase Triad3A Negatively Regulates the RIG-I/MAVS Signaling Pathway by Targeting TRAF3 for Degradation by Peyman Nakhaei (258397)

“…Triad3A was induced following dsRNA exposure or virus infection and decreased TRAF3 levels in a dose-dependent manner; moreover, Triad3A expression blocked IRF-3 activation by Ser-396 phosphorylation and inhibited the expression of type 1 interferon and antiviral genes. …”

Image 3_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image_3_Social Deficits and Cerebellar Degeneration in Purkinje Cell Scn8a Knockout Mice.JPEG by Xiaofan Yang (767342)

“…At 9 months of age, Scn8a^flox/flox, L7Cre⁺ mice exhibited decreased cerebellar size and a reduced number of cerebellar Purkinje cells more profoundly, with evidence of additional neurodegeneration in the molecular layer and deep cerebellar nuclei. …”

Table_1_Social Deficits and Cerebellar Degeneration in Purkinje Cell Scn8a Knockout Mice.XLSX by Xiaofan Yang (767342)

“…At 9 months of age, Scn8a^flox/flox, L7Cre⁺ mice exhibited decreased cerebellar size and a reduced number of cerebellar Purkinje cells more profoundly, with evidence of additional neurodegeneration in the molecular layer and deep cerebellar nuclei. …”

Image_2_Social Deficits and Cerebellar Degeneration in Purkinje Cell Scn8a Knockout Mice.JPEG by Xiaofan Yang (767342)

“…At 9 months of age, Scn8a^flox/flox, L7Cre⁺ mice exhibited decreased cerebellar size and a reduced number of cerebellar Purkinje cells more profoundly, with evidence of additional neurodegeneration in the molecular layer and deep cerebellar nuclei. …”

Image 4_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image_1_Social Deficits and Cerebellar Degeneration in Purkinje Cell Scn8a Knockout Mice.JPEG by Xiaofan Yang (767342)

“…At 9 months of age, Scn8a^flox/flox, L7Cre⁺ mice exhibited decreased cerebellar size and a reduced number of cerebellar Purkinje cells more profoundly, with evidence of additional neurodegeneration in the molecular layer and deep cerebellar nuclei. …”

Data Sheet 1_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.pdf by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image 1_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Table 1_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.docx by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image 5_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image 6_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image 2_ATP/P2X7 receptor signal aggravates ischemic stroke injury by activating Th17 cells via STAT3/IL-21 pathway.tif by Wenying Liu (781302)

“…Rag2^-/- mice that received P2X7-KO CD4⁺T cells demonstrated reduced ischemic-reperfusion injury and a decreased level of IL-17A and frequency of Th17 cells compared to Rag2^-/- mice that received wild type CD4⁺T cells. …”

Image_4_Social Deficits and Cerebellar Degeneration in Purkinje Cell Scn8a Knockout Mice.JPEG by Xiaofan Yang (767342)

“…At 9 months of age, Scn8a^flox/flox, L7Cre⁺ mice exhibited decreased cerebellar size and a reduced number of cerebellar Purkinje cells more profoundly, with evidence of additional neurodegeneration in the molecular layer and deep cerebellar nuclei. …”

Data_Sheet_1_CagA+Helicobacter pylori, Not CagA–Helicobacter pylori, Infection Impairs Endothelial Function Through Exosomes-Mediated ROS Formation.pdf by Xiujuan Xia (11929241)

“…CagA⁺H. pylori, not CagA^–H. pylori, infection significantly increased aortic ROS production, decreased ACh-induced aortic relaxation, and enhanced early atherosclerosis formation, which were prevented with N-acetylcysteine treatment. …”