The landscape of A-to-I editomes in <i>D</i>. <i>melanogaster</i>. by Yuange Duan (3829594)

“…<p>(A) A flowchart of A-to-I editing detection in brains of <i>D</i>. …”

HUK up-regulated pro-angiogeic factors in ERK1/2 dependent way in cultured endothelial cells. by Lijuan Han (553897)

“…(G) ELISA was used to measure apelin concentration in cultured cell medium. (H) F13A decreased VEGF expression up- regulated by HUK. …”

Table1_Influence of FMO3 and CYP3A4 Polymorphisms on the Pharmacokinetics of Teneligliptin in Humans.docx by Jin-Woo Park (624549)

“…However, the C_max values significantly decreased but the AUC values did not significantly vary in subjects with CYP3A4 polymorphism (rs2242480). …”

The cholesterol biosynthesis pathway. by Jonathon D. Klein (679267)

“…Blue circles denote genes with a fold change greater than −1.3, while green denote those less than −1.3. …”

Table3_Influence of FMO3 and CYP3A4 Polymorphisms on the Pharmacokinetics of Teneligliptin in Humans.docx by Jin-Woo Park (624549)

“…However, the C_max values significantly decreased but the AUC values did not significantly vary in subjects with CYP3A4 polymorphism (rs2242480). …”

Table2_Influence of FMO3 and CYP3A4 Polymorphisms on the Pharmacokinetics of Teneligliptin in Humans.docx by Jin-Woo Park (624549)

“…However, the C_max values significantly decreased but the AUC values did not significantly vary in subjects with CYP3A4 polymorphism (rs2242480). …”

Image1_Influence of FMO3 and CYP3A4 Polymorphisms on the Pharmacokinetics of Teneligliptin in Humans.tiff by Jin-Woo Park (624549)

“…However, the C_max values significantly decreased but the AUC values did not significantly vary in subjects with CYP3A4 polymorphism (rs2242480). …”

Confirmation of altered expression by qRT-PCR in functional pathway genes. by Jonathon D. Klein (679267)

“…<i>Srebf1</i>, a transcriptioin factor that activates many genes in the synthesis pathway, was significantly decreased in both analyses. …”

Pharmacological inhibitors of signaling pathways decrease CT-1-induced MMP-1 gene expression. by Akinori Tokito (436385)

Glycolytic type 2x, but not type 2b, fibers decrease following muscle regeneration. by Steven J. Foltz (845135)

NO donors and Bradykinin increase 26S proteasome reporter protein levels and decrease 26S proteasome activity. by Hongtao Liu (114450)

Table3_SLC38A10 Regulate Glutamate Homeostasis and Modulate the AKT/TSC2/mTOR Pathway in Mouse Primary Cortex Cells.XLSX by Rekha Tripathi (9092534)

“…SLC38A10 is a member of the SLC38 family and regulates protein synthesis and cellular stress responses. …”

HOTAIR mediates cisplatin resistance in nasopharyngeal carcinoma by regulating miR-106a-5p/SOX4 axis by Wei Cao (50866)

“…MiR-106a-5p targets HOTAIR and SOX4; thus, silencing of HOTAIR significantly increased miR-106a-5p expression. …”

Table2_SLC38A10 Regulate Glutamate Homeostasis and Modulate the AKT/TSC2/mTOR Pathway in Mouse Primary Cortex Cells.XLSX by Rekha Tripathi (9092534)

“…SLC38A10 is a member of the SLC38 family and regulates protein synthesis and cellular stress responses. …”

Video_2_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_5_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_4_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_3_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_6_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_1_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”