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largest decrease » largest decreases (Expand Search), marked decrease (Expand Search)
larger decrease » marked decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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23461
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23462
Data_Sheet_1_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.pdf
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23463
Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23464
Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23465
Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23466
Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23467
Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23468
Data_Sheet_1_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.pdf
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23469
Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23470
Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23471
Data_Sheet_5_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23472
Data_Sheet_5_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23473
Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx
Published 2021“…P16<sup>INK4a</sup> deletion could maintain barrier function and microbiota balance in Bmi-1<sup>–/–</sup> mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”
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23474
Image_5_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23475
Image_4_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23476
Image_6_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23477
Table_1_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.xlsx
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23478
Table_3_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.docx
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23479
Image_7_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”
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23480
Image_1_Midkine-a Regulates the Formation of a Fibrotic Scar During Zebrafish Heart Regeneration.JPEG
Published 2021“…Analysis of injured hearts showed that loss of mdka decreased endothelial cell proliferation and resulted in an arrest in heart regeneration characterized by retention of a collagenous scar. …”