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Showing 9,461 - 9,480 results of 52,235 for search '(( a ((mean decrease) OR (linear decrease)) ) OR ( a ((largest decrease) OR (larger decrease)) ))', query time: 0.84s Refine Results
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    Decreased clonogenic capacity of U87MG and U251MG GB cells following treatment with SHG-44 and cis-platin at equal concentrations. by Denis Mustafov (19137870)

    Published 2025
    “…<p><b>(A)</b> A significant reduction in colony-forming ability was observed in U87MG cells treated with 100ΜM SHG-44 compared to 100ΜM cis-platin and the untreated control. …”
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  16. 9476

    Simultaneously inhibition <i>Nlu-miR-34</i> and <i>NlInR2</i> in SW strain induced the decrease of SW BPHs. by Xinhai Ye (6857771)

    Published 2019
    “…<p>(A) Antagomir-34 (with two different quantities) and ds<i>NlInR2</i> (0.84 ng) were injected into the third instar SW strain nymphs, showed that injection of antagomir-34 decreased the ratio of SW type BPH (Chi-square test, 40ng: χ<sup>2</sup> = 5.8, df = 1, <i>p</i> = 0.003; 60ng: χ<sup>2</sup> = 7.9, df = 1, <i>p</i> = 0.042). …”
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    A H<sub>2</sub>S-Nampt Dependent Energetic Circuit Is Critical to Survival and Cytoprotection from Damage in Cancer Cells by Reiko Sanokawa-Akakura (254909)

    Published 2014
    “…Together, these data show that upon recovery from potential lethal damage, H<sub>2</sub>S-Nampt directs energy expenditure and aerobic glycolysis in cancer cells, leads to their exponential growth, and causes a high degree of tolerance to damage. Identification of H<sub>2</sub>S-Nampt as a pathway responsible for induction of damage tolerance in cancer cells may underlie resistance to therapy and offers the opportunity to target this pathway as a means in treatment of cancer.…”