Table_5_A Comparative Study of the Gut Microbiota Associated With Immunoglobulin a Nephropathy and Membranous Nephropathy.XLSX by Ruijuan Dong (751134)

“…<p>The pathogenesis of immunoglobulin A nephropathy (IgAN) and membranous nephropathy (MN) is characterized by immune dysregulation, which is related to gut dysbiosis. …”

Structural Insight into and Mutational Analysis of Family 11 Xylanases: Implications for Mechanisms of Higher pH Catalytic Adaptation by Wenqin Bai (767975)

“…<div><p>To understand the molecular basis of higher pH catalytic adaptation of family 11 xylanases, we compared the structures of alkaline, neutral, and acidic active xylanases and analyzed mutants of xylanase Xyn11A-LC from alkalophilic <i>Bacillus</i> sp. SN5. …”

Table_4_A Comparative Study of the Gut Microbiota Associated With Immunoglobulin a Nephropathy and Membranous Nephropathy.XLSX by Ruijuan Dong (751134)

“…<p>The pathogenesis of immunoglobulin A nephropathy (IgAN) and membranous nephropathy (MN) is characterized by immune dysregulation, which is related to gut dysbiosis. …”

MiR-15a-5p Knockdown up-Regulated ABCB1 Expression and Abated HNSCC Progression via the NF-κB Signaling Pathway by Jing Liu (38537)

“…MiR-15a-5p was a regulator for ABCB1, which was up-regulated after miR-15a-5p inhibition <i>in vitro</i>. …”

Data_Sheet_1_LolA and LolB from the plant-pathogen Xanthomonas campestris forms a stable heterodimeric complex in the absence of lipoprotein.PDF by Valentina Furlanetto (16530921)

“…Earlier studies have shown that loss of Xcc LolA or LolB leads to decreased virulence and pathogenicity during plant infection, which motivates studies to better understand the Lol system in Xcc. …”

DataSheet_1_Gut Microbiome Characteristics in IgA Nephropathy: Qualitative and Quantitative Analysis from Observational Studies.docx by Shisheng Han (10019855)

“…Background<p>Recent data indicate the importance of gut-kidney axis in the pathogenesis of Immunoglobulin A nephropathy (IgAN). Growing evidence suggests the alterations of diversity and composition of gut microbiome among patients with IgAN, however, the details are not yet fully understood.…”

Functional Characterization of the Receiver Domain for Phosphorelay Control in Hybrid Sensor Kinases by Emiko Kinoshita-Kikuta (766663)

“…However, both wild-type EvgS and the Ala-substituted mutant of His in the HPt domain showed a phosphorylation ratio of less than 25%, which gradually decreased after 10 min. …”

DataSheet_1_Interplays of mutations in waaA, cmk, and ail contribute to phage resistance in Yersinia pestis.docx by Lisheng Xiao (15919517)

“…The waaA* mutation leads to decreased phage adsorption because of the failure to synthesize the lipopolysaccharide core. …”

Table_7_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.xlsx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

Table_1_MEF2A Is the Trigger of Resveratrol Exerting Protection on Vascular Endothelial Cell.XLS by Benrong Liu (200777)

“…Silencing MEF2A in VE of apoE^−/− mice decreased the expression of SIRT1, increased the plasma LDL-c, and abrogated the function of resveratrol on reducing triglyceride. …”

Table_8_MEF2A Is the Trigger of Resveratrol Exerting Protection on Vascular Endothelial Cell.DOCX by Benrong Liu (200777)

“…Silencing MEF2A in VE of apoE^−/− mice decreased the expression of SIRT1, increased the plasma LDL-c, and abrogated the function of resveratrol on reducing triglyceride. …”

Table_3_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.xlsx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

Table_8_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.xlsx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

Table_4_MEF2A Is the Trigger of Resveratrol Exerting Protection on Vascular Endothelial Cell.XLS by Benrong Liu (200777)

“…Silencing MEF2A in VE of apoE^−/− mice decreased the expression of SIRT1, increased the plasma LDL-c, and abrogated the function of resveratrol on reducing triglyceride. …”

Table_4_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.xlsx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

DataSheet_1_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.docx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

A Possible Link between Food and Mood: Dietary Impact on Gut Microbiota and Behavior in BALB/c Mice by Bettina Pyndt Jørgensen (617023)

“…<div><p>Major depressive disorder is a debilitating disease in the Western World. A western diet high in saturated fat and refined sugar seems to play an important part in disease development. …”

Table_2_Pathogenesis of Enamel-Renal Syndrome Associated Gingival Fibromatosis: A Proteomic Approach.xlsx by Victor Simancas Escorcia (9741794)

“…<p>The enamel renal syndrome (ERS) is a rare disorder featured by amelogenesis imperfecta, gingival fibromatosis and nephrocalcinosis. …”

Table_3_MEF2A Is the Trigger of Resveratrol Exerting Protection on Vascular Endothelial Cell.DOCX by Benrong Liu (200777)

“…Silencing MEF2A in VE of apoE^−/− mice decreased the expression of SIRT1, increased the plasma LDL-c, and abrogated the function of resveratrol on reducing triglyceride. …”

Table_9_MEF2A Is the Trigger of Resveratrol Exerting Protection on Vascular Endothelial Cell.XLSX by Benrong Liu (200777)

“…Silencing MEF2A in VE of apoE^−/− mice decreased the expression of SIRT1, increased the plasma LDL-c, and abrogated the function of resveratrol on reducing triglyceride. …”