A simplified SCOPE score. by Ismael Francisco Aomar-Millán (14838595)

Increased Von Willebrand factor, decreased ADAMTS13 and thrombocytopenia in melioidosis - Fig 2 by Emma Birnie (3449603)

Increased Von Willebrand factor, decreased ADAMTS13 and thrombocytopenia in melioidosis - Fig 3 by Emma Birnie (3449603)

Increased Von Willebrand factor, decreased ADAMTS13 and thrombocytopenia in melioidosis - Fig 4 by Emma Birnie (3449603)

Left ventricular end diastolic diameter by Biagio Castaldi (20673392)

NCM exposure increases hepatic creatine kinase activity and total ATPase activity, which is associated with decreases in hepatic ABCA1, CD36, and L-FABP protein expression and circ... by Ryan J. Mailloux (50825)

Dr. Hannah Lapp’s—like custom features in action. by Piotr Popik (292215)

Data_Sheet_1_A Glycosylphosphatidylinositol-Anchored α-Amylase Encoded by amyD Contributes to a Decrease in the Molecular Mass of Cell Wall α-1,3-Glucan in Aspergillus nidulans.doc... by Ken Miyazawa (2886026)

“…<p>α-1,3-Glucan is one of the main polysaccharides in the cell wall of Aspergillus nidulans. …”

Data_Sheet_1_A Glycosylphosphatidylinositol-Anchored α-Amylase Encoded by amyD Contributes to a Decrease in the Molecular Mass of Cell Wall α-1,3-Glucan in Aspergillus nidulans.doc... by Ken Miyazawa (2886026)

“…<p>α-1,3-Glucan is one of the main polysaccharides in the cell wall of Aspergillus nidulans. …”

Ovariectomy (OVX) induced decrease in hexokinase expression and activity: prevention by 17β-estradiol. by Fan Ding (302565)

“…<p>A. In female 3×TgAD brains, OVX induced a significant decrease in protein expression of hexokinase 2 (*, P<0.05, bars represent mean value±SEM, n = 5−6), which was partially prevented by E2 treatment. …”

Image5_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image2_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image4_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image7_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image6_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image1_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

Image3_PKA inhibition is a central step in D,L-methadone-induced ER Ca2+ release and subsequent apoptosis in acute lymphoblastic leukemia.JPEG by Hamza Kamran (18433152)

“…The 14–22 amide (myr) PKA inhibitor alone elicits ER Ca²⁺ release, and subsequent treatment with D,L-methadone does not cause additional ER Ca²⁺ release, indicating that PKA inhibition is a key step in D,L-methadone-induced ER Ca²⁺ release and can bypass the D,L-methadone-OPRM1-AC-cAMP step. …”

AMPs are increased in lesional HS, but DCD and other sweat-gland associated proteins are decreased. by Margaret Coates (6058580)

“…Genes highlighted in blue are downregulated genes of interest; genes highlighted in red are upregulated genes of interest. While <i>DCD</i> is downregulated in HS lesional skin, many other AMPs and interferon-associated molecules are enriched in lesional HS. …”

The levels of antitoxin and toxin-antitoxin complexes decrease with increasing number of operator binding sites. by Lendert Gelens (451747)

Typical tracing of the effect of i.a. endothelin 0.5 10⁻⁵ M on arterial pressure in rats: the initial transient decrease of arterial pressure is followed by a sustained... by Matthieu Roustit (315980)