Zinc exposure attenuates NR2A clusters and currents. by Jia Zhu (135506)

“…(B) and (C) show the quantification data of (A). Both the number and mean intensity of NR2A clusters significantly decreased after zinc treatment. …”

Table 1_Tissue-specific effects of bacterial PncA overexpression on NAD+ metabolism and aging in mice: implications for tissue-specific aging interventions.docx by Fengjiao Huo (15993086)

“…Overexpressing the bacterial nicotinamidase PncA in mice increased NAD⁺ levels in the liver and kidneys but decreased levels in the heart and hippocampus. …”

Table 1_Epidemiology and genetic characteristics of coxsackievirus A16 associated with hand-foot-mouth disease in Hangzhou city, Zhejiang province from 2021 to 2024.docx by Qihao Xu (5513795)

“…The detected number of CV-A16 began to rise in March, peaked in May–August, and then decreased in September. …”

Retinol and mineral status in grazing foals during the dry season by Maria Lindomárcia Leonardo da Costa (12802135)

“…Beta-carotene in grass showed a downward trend over time from 38.53 to 0.09 mg/kg of dry matter. …”

Data Sheet 1_Semaglutide attenuates myocardial ischemia-reperfusion injury by inhibiting ferroptosis of cardiomyocytes via activation of PKC-S100A9 axis.pdf by Yan Liu (25061)

“…In vitro, experiments showed that semaglutide decreased the expression of S100A9 by activating the Protein Kinase C(PKC) pathway, thus inhibiting ferroptosis in cardiomyocytes.…”

Data Sheet 2_Semaglutide attenuates myocardial ischemia-reperfusion injury by inhibiting ferroptosis of cardiomyocytes via activation of PKC-S100A9 axis.docx by Yan Liu (25061)

“…In vitro, experiments showed that semaglutide decreased the expression of S100A9 by activating the Protein Kinase C(PKC) pathway, thus inhibiting ferroptosis in cardiomyocytes.…”

Table 1_Semaglutide attenuates myocardial ischemia-reperfusion injury by inhibiting ferroptosis of cardiomyocytes via activation of PKC-S100A9 axis.docx by Yan Liu (25061)

“…In vitro, experiments showed that semaglutide decreased the expression of S100A9 by activating the Protein Kinase C(PKC) pathway, thus inhibiting ferroptosis in cardiomyocytes.…”

Image 1_Epidemiology and genetic characteristics of coxsackievirus A16 associated with hand-foot-mouth disease in Hangzhou city, Zhejiang province from 2021 to 2024.jpeg by Qihao Xu (5513795)

“…The detected number of CV-A16 began to rise in March, peaked in May–August, and then decreased in September. …”

Efmoroctocog Alfa: A Review in Haemophilia A by James E. Frampton (4995257)

“…</p> <p><b> </b></p> <p><b>Authorship and Conflict of interest</b> James E. Frampton is a salaried employee of Adis International Ltd/Springer Nature, and declares no relevant conflicts of interest. …”

Porcine Coro1A inhibit <i>H.parasuis</i> induced NF-κB activation. by Chong Liu (450204)

“…<p>(A) PK-15 cells were transfected with pNF-κB-luc (0.2 µg) and pRL-TK (0.05 µg), then cells were control-inoculated, live or heat-killed <i>H.parasuis</i> (10⁷ CFU) inoculated. …”

Reduction of NF-κB activation when porcine Coro1A was overexpressed. by Chong Liu (450204)

“…<p>(A) PK-15 cells were co-transfected with the pNF-κB-luc reporter plasmid (0.2 µg), pRL-TK plasmid (0.05 µg), along with 0.6 µg of the expression plasmid encoding porcine Coro1A protein. …”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Mechanism Studies on the Chemical Stability of FIrpic, a Typical Blue Phosphorescent Emitter for Electroluminescence, in the Redox States by Weiqiang Wei (19728558)

“…Charge analysis indicates a decreasing trend in electronic delocalization between the LP_N electron donor natural bond orbital (NBO) and the d*<sub>N–Ir(pic.)…”

Allosteric Regulation of IGF2BP1 as a Novel Strategy for the Activation of Tumor Immune Microenvironment by Yang Liu (4829)

“…However, currently, the biological function of IGF2BP1 in regulating the TIME is not well-understood. In this study, we report that IGF2BP1 knockdown induces cancer cell apoptosis, thereby significantly not only activating immune cell infiltration including CD4⁺, CD8⁺ T cells, CD56⁺ NK cells, and F4/80⁺ macrophage but also decreasing PD-L1 expression in hepatocellular carcinoma (HCC). …”

Allosteric Regulation of IGF2BP1 as a Novel Strategy for the Activation of Tumor Immune Microenvironment by Yang Liu (4829)

“…However, currently, the biological function of IGF2BP1 in regulating the TIME is not well-understood. In this study, we report that IGF2BP1 knockdown induces cancer cell apoptosis, thereby significantly not only activating immune cell infiltration including CD4⁺, CD8⁺ T cells, CD56⁺ NK cells, and F4/80⁺ macrophage but also decreasing PD-L1 expression in hepatocellular carcinoma (HCC). …”

Allosteric Regulation of IGF2BP1 as a Novel Strategy for the Activation of Tumor Immune Microenvironment by Yang Liu (4829)

“…However, currently, the biological function of IGF2BP1 in regulating the TIME is not well-understood. In this study, we report that IGF2BP1 knockdown induces cancer cell apoptosis, thereby significantly not only activating immune cell infiltration including CD4⁺, CD8⁺ T cells, CD56⁺ NK cells, and F4/80⁺ macrophage but also decreasing PD-L1 expression in hepatocellular carcinoma (HCC). …”