App user onboarding trends. by Henri Claude Moungui (22227043)

“…</p><p>Methods</p><p>We conducted a pilot quasi‑experimental before‑and‑after study comparing three 6‑month phases—pre‑campaign, campaign, and post‑campaign. …”

App user base growth trends. by Henri Claude Moungui (22227043)

“…</p><p>Methods</p><p>We conducted a pilot quasi‑experimental before‑and‑after study comparing three 6‑month phases—pre‑campaign, campaign, and post‑campaign. …”

Data_sheet_1_Mangrove Loss and Gain in a Densely Populated Urban Estuary: Lessons From the Guangdong-Hong Kong-Macao Greater Bay Area.docx by Heng Wang (76752)

“…<p>Understanding the recent changes in mangrove adjacent to mega-cities is critical for conservation, management, and policymaking in coastal zones with fast population growth and global change. Here we investigated mangrove area changes in one of the world’s largest urban areas near the main estuaries in the Guangdong-Hong Kong-Macao Greater Bay Area (GBA). …”

Image_1_Involvement of the Precuneus/Posterior Cingulate Cortex Is Significant for the Development of Alzheimer’s Disease: A PET (THK5351, PiB) and Resting fMRI Study.TIFF by Takamasa Yokoi (5821856)

“…All participants underwent resting state functional MRI and ¹⁸F-THK5351 PET scans. We used scaled subprofile modeling/principal component analysis (SSM/PCA) to reduce the complexity of multivariate data and to identify patterns that exhibited the largest statistical effects (variances) in THK5351 concentration in AD and healthy controls.…”

Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide by Yakov M. Koen (1970593)

“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”

Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide by Yakov M. Koen (1970593)

“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”

Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide by Yakov M. Koen (1970593)

“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”

Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide by Yakov M. Koen (1970593)

“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”

Literature search and screening process. by Xiaohong Mai (19338585)

“…<div><p>Increasing evidence has shown that gut microbiota (GM) was involved in the pathophysiology of musculoskeletal disorders through multiple pathways such as protein anabolism, chronic inflammation and immunity, and imbalanced metabolism. We performed a systematic review and meta-analysis of human studies to evaluate GM diversity differences between individuals with and without sarcopenia, and explore bacteria with potential to become biomarkers. …”

Sampling locations for banana aphids. by Aime Cheoh Enoh (20141758)

“…Four isolates (BIITAC6.2.2, BIITAC10.3.3, BIITAC8.1.5, and MIITAC6.2.2) were highly pathogenic, causing greater than 75% aphid mortality in all populations. A significant decrease in aphid fecundity was observed with BIITAC6.2.2, MIITAC6.2.2, and BIITAC10.3.3. …”

Mean value of the longitudinal scaling exponent as a function of the growth rate threshold <i>b</i>_<i>c</i>. by Fabiano L. Ribeiro (4893388)

“…The mean value of decreases drastically as <i>b</i>_<i>c</i> increases. …”

Microbial ecology and biogeochemistry of hypersaline sediments in Orca Basin by Lisa M. Nigro (8741892)

“…Illumina and clone library surveys consistently detected MG-I Thaumarchaeota and halotolerant Deltaproteobacteria in the hypersaline anoxic sediments, but relative abundances of the KB1 lineage differed between the two sequencing methods. …”

Flowchart of the study population. by Anders H. Hjulmand (18476019)

“…Mean test scores varied by demographic and socioeconomic characteristics, most notably with education and labour market affiliation of parents. For every 1-point decrease in the test scores, there was a 0.95% (95% CI: 0.93%; 0.97%) lower probability of scoring B or higher in the ninth-grade exam and a 1.03% (95% CI: 1.00%; 1.05%) lower probability of completing high school within five years after graduating from lower secondary school. …”

Video_2_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.MP4 by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Video_1_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.MP4 by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Data_Sheet_1_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.PDF by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Data_Sheet_1_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.PDF by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Video_1_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.MP4 by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Video_2_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.MP4 by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”

Video_2_NudC L279P Mutation Destabilizes Filamin A by Inhibiting the Hsp90 Chaperoning Pathway and Suppresses Cell Migration.MP4 by Min Liu (45756)

“…However, the regulatory mechanism of filamin A stability remains unclear. Here, we find that nuclear distribution gene C (NudC), a cochaperone of heat shock protein 90 (Hsp90), is required to stabilize filamin A in mammalian cells. …”