Showing 16,481 - 16,500 results of 132,736 for search '(( a e decrease ) OR ( 5 ((point decrease) OR (((mean decrease) OR (a decrease)))) ))', query time: 1.99s Refine Results
  1. 16481

    Image5_Analysis of Geometric and Hemodynamic Profiles in Rat Arteriovenous Fistula Following PDE5A Inhibition.tif by Hannah Northrup (5938436)

    Published 2021
    “…Geometrical parameters include arterial-venous (A-V) distance, anastomosis angle, tortuosity, and nonplanarity angle magnitude. …”
  2. 16482
  3. 16483
  4. 16484

    S1 Data - by Kadio Jean-Jacques Olivier Kadio (15503159)

    Published 2023
    “…Retention 12 months after ART initiation was 48.7% (95%CI 48.1–49.4%). The LTFU rate was 54.5 per 1000 person-months (95% CI 53.6–55.4), with the peak hazards of LTFU occurring after the first visit and decreasing steadily over time. …”
  5. 16485

    Participant characteristics. by Kadio Jean-Jacques Olivier Kadio (15503159)

    Published 2023
    “…Retention 12 months after ART initiation was 48.7% (95%CI 48.1–49.4%). The LTFU rate was 54.5 per 1000 person-months (95% CI 53.6–55.4), with the peak hazards of LTFU occurring after the first visit and decreasing steadily over time. …”
  6. 16486
  7. 16487
  8. 16488
  9. 16489
  10. 16490
  11. 16491
  12. 16492
  13. 16493
  14. 16494
  15. 16495

    Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
  16. 16496

    Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
  17. 16497

    Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
  18. 16498

    Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
  19. 16499

    Image_3_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
  20. 16500

    Image_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”