Showing 1,161 - 1,180 results of 104,576 for search '(( a fold decrease ) OR ( 5 ((((nn decrease) OR (point decrease))) OR (a decrease)) ))', query time: 1.56s Refine Results
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    Overview of study procedures. by Matthew G. Vinson (22593374)

    Published 2025
    “…VIDEO and CONTROL were associated with a similar rise in intent to decrease OTC NSAID use (1.92 (SD: 4.41) vs. 1.36 (SD: 3.46), p = 0.150) and a similar decrease in NSAIDs exposure (−32.8% in VIDEO and −36.5% in CONTROL, p = 0.520) 4 weeks post-intervention. …”
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    The BDNF-evoked neuronal IEG response is counteracted by ephrin-A5 co-application. by Christin Meier (201634)

    Published 2011
    “…At this extended exposure (4h) to both guidance cues, co-application of ephrin-A5 failed to decrease BDNF-stimulated gene expression. …”
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    Adsorption characteristics of the carbonaceous adsorbents for organic compounds in a model exhaust gas from thermal treatment processing by Katsuya Kawamoto (12369640)

    Published 2022
    “…The experimental results showed the following: 1) The adsorption capacity of every carbonaceous material for organic vapor decreased with increasing temperature, with roughly a three-fold difference in the capacity, between 150°C and 190°C in the dry gas. …”
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    A Peptide Mimetic of 5-Acetylneuraminic Acid-Galactose Binds with High Avidity to Siglecs and NKG2D by Laura L. Eggink (760403)

    Published 2015
    “…Using plant lectins as analogs of lectin-type cell-surface receptors, a tetravalent form of a peptide with the sequence NPSHPLSG, designated svH1C, bound with high avidity to lectins specific for glycans with terminal 5-acetylneuraminic acid (Neu5Ac)-galactose (Gal)/N-acetylgalactosamine (GalNAc) sequences. …”
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    Molecular insights in the pathogenesis of classical Ehlers-Danlos syndrome from transcriptome-wide expression profiling of patients’ skin fibroblasts by Nicola Chiarelli (756982)

    Published 2019
    “…<div><p>Classical Ehlers-Danlos syndrome (cEDS) is a dominant inherited connective tissue disorder mainly caused by mutations in the <i>COL5A1</i> and <i>COL5A2</i> genes encoding type V collagen (COLLV), which is a fibrillar COLL widely distributed in a variety of connective tissues. cEDS patients suffer from skin hyperextensibility, abnormal wound healing/atrophic scars, and joint hypermobility. …”
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