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non decrease » nn decrease (Expand Search), point decrease (Expand Search), note decreased (Expand Search)
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38141
Image_4_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.TIF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38142
Data_Sheet_3_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.PDF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38143
Data_Sheet_2_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.PDF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38144
Image_3_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.TIF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38145
Image_2_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.TIF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38146
Data_Sheet_1_Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis.PDF
Published 2020“…With the wild-type cell line serving as an isogenic control and MNs from a patient-derived iPSC line with an SOD1-A4V mutation as a comparator, we identified pathological phenotypes relevant to ALS. …”
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38147
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38148
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38149
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38150
Unraveling endothelin-1 induced hypercontractility of human pulmonary artery smooth muscle cells from patients with pulmonary arterial hypertension
Published 2018“…Treatment of HPASMC with vasoactive peptides, endothelin-1 (ET-1) and bradykinin (BK) but not angiotensin II, induced a temporal decrease in the electrical impedance profile mirroring constrictive morphological change of the cells which typically was more robust in PAH as opposed to non-PAH cells. …”
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38151
FIGURE 4 from Targeting <i>MALAT1</i> Augments Sensitivity to PARP Inhibition by Impairing Homologous Recombination in Prostate Cancer
Published 2023“…<b>F,</b> Flow cytometry–based apoptosis assay using Annexin V-PE and 7-AAD staining in the same cells as in A. …”
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38152
The Y-Box Binding Protein 1 Suppresses Alzheimer’s Disease Progression in Two Animal Models
Published 2015“…YB-1-treated OBX and 5XFAD mice showed a decreased level of brain β-amyloid. …”
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38153
The complementary chimera IIS4I shifts the gating of Ca<sub>V</sub>1.2 channels towards low-voltage behavior.
Published 2018“…Note that chimeric channels availability decreases proportionally as the <i>V</i><sub>m</sub> become more positive, similar to the behavior of Ca<sub>V</sub>3.3-WT channels (see <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0193490#pone.0193490.g004" target="_blank">Fig 4</a>). …”
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38154
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38155
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38156
Image_2_Nexmifa Regulates Axon Morphogenesis in Motor Neurons in Zebrafish.JPEG
Published 2022“…In particular, when compared with other pathways, DEGs were highest (84) in the axon guidance pathway, according to Organismal Systems. Efna5b, bmpr2b, and sema6ba were decreased markedly in nexmifa-depleted zebrafish embryos. …”
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38157
Table_2_Nexmifa Regulates Axon Morphogenesis in Motor Neurons in Zebrafish.XLS
Published 2022“…In particular, when compared with other pathways, DEGs were highest (84) in the axon guidance pathway, according to Organismal Systems. Efna5b, bmpr2b, and sema6ba were decreased markedly in nexmifa-depleted zebrafish embryos. …”
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38158
Image_1_Nexmifa Regulates Axon Morphogenesis in Motor Neurons in Zebrafish.JPEG
Published 2022“…In particular, when compared with other pathways, DEGs were highest (84) in the axon guidance pathway, according to Organismal Systems. Efna5b, bmpr2b, and sema6ba were decreased markedly in nexmifa-depleted zebrafish embryos. …”
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38159
Table_1_Nexmifa Regulates Axon Morphogenesis in Motor Neurons in Zebrafish.DOCX
Published 2022“…In particular, when compared with other pathways, DEGs were highest (84) in the axon guidance pathway, according to Organismal Systems. Efna5b, bmpr2b, and sema6ba were decreased markedly in nexmifa-depleted zebrafish embryos. …”
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38160
Image_3_Nexmifa Regulates Axon Morphogenesis in Motor Neurons in Zebrafish.JPEG
Published 2022“…In particular, when compared with other pathways, DEGs were highest (84) in the axon guidance pathway, according to Organismal Systems. Efna5b, bmpr2b, and sema6ba were decreased markedly in nexmifa-depleted zebrafish embryos. …”